Fei Hu
Tongji University
6 Papers
Fei Hu is an academic researcher from Tongji University. The author has contributed to research in topics: Autophagy & Programmed cell death. The author has an hindex of 6, co-authored 6 publications. Previous affiliations of Fei Hu include Second Military Medical University.
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Papers
Targeting autophagy potentiates chemotherapy-induced apoptosis and proliferation inhibition in hepatocarcinoma cells
Xianling Guo,Ding Li,Fei Hu,Jianrui Song,Shanshan Zhang,Wei-jie Deng,Kai Sun,Qiudong Zhao,Xuqin Xie,Yu-jiao Song,Mengchao Wu,Lixin Wei +11 more
TL;DR: It is suggested that autophagy is a potential novel target to improve therapy efficiency of conventional chemotherapeutics towards HCC.
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Docetaxel-mediated autophagy promotes chemoresistance in castration-resistant prostate cancer cells by inhibiting STAT3.
TL;DR: It is shown that autophagy is triggered during chemotherapy and that inhibiting Autophagy increased chemosensitivity of castration-resistant prostate cancer (CRPC) cells, and results suggest that STAT3 contributes to CRPC cell survival and chemoresistance by modulatingAutophagy.
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Inhibition of p53 increases chemosensitivity to 5-FU in nutrient-deprived hepatocarcinoma cells by suppressing autophagy
Xianling Guo,Fei Hu,Fei Hu,Shanshan Zhang,Qiudong Zhao,Chen Zong,Fei Ye,Shiwei Guo,Jian-wei Zhang,Rong Li,Mengchao Wu,Lixin Wei +11 more
TL;DR: The results suggest that p53 contributes to cell survival and chemoresistance in HCC under nutrient-deprived conditions by modulating autophagy activation.
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Inhibition of autophagy in hepatocarcinoma cells promotes chemotherapeutic agent-induced apoptosis during nutrient deprivation.
TL;DR: The present study suggests that induction of autophagy confers a survival advantage for hepatocarcinoma cells during nutrient deprivation, not only rescuing cells from nutrient deficiency-induced cell apoptosis, but also protecting cells from chemotherapy- induced cell death.
Suppression of p53 potentiates chemosensitivity in nutrient‑deprived cholangiocarcinoma cells via inhibition of autophagy
TL;DR: It is demonstrated that the inhibition of p53 enhanced the sensitivity to chemotherapeutic agents in nutrient-deprived cholangiocarcinoma cells, and thus contributes to cell survival and chemoresistance.