About: Dieting is a research topic. Over the lifetime, 3526 publications have been published within this topic receiving 176826 citations. The topic is also known as: diet & going on a diet.
TL;DR: In this article , the immediate sex-specific effects of transitioning to a high-fat diet in C57BL/6J mice as well as monitored whether these effects are altered after sustained HFD feeding and whether sex affects the response to a return to chow, representative of dieting.
Abstract: Female mice are often excluded from diet-induced obesity studies as they are more resistant to the obesifying effects of a high-fat diet (HFD). However, the underlying mechanisms behind this sex disparity may actually have important implications for the development and management of obesity in humans. Therefore, we systematically investigated the immediate sex-specific effects of transitioning to a HFD in C57BL/6J mice as well as monitored whether these effects are altered after sustained HFD feeding and whether sex affects the response to a return to chow, representative of dieting.Dual X-ray absorptiometry (DXA) analysis of body composition, indirect calorimetry measurements, and qPCR analysis of hypothalamic and brainstem regions were performed on male and female C57BL/6J mice.HFD had immediate and dramatic effects in males, increasing fat mass by 58% in the first 3 days. The resistance to the obesifying effect of HFD in females was linked both to an ability to maintain activity levels as well as to an immediate and significantly enhanced reduction in respiratory quotient (RQ), suggesting a greater ability to utilise fat in the diet as a source of fuel. Mechanistically, this sex disparity may be at least partially due to inherent sex differences in the catabolic (POMC/CART) versus anabolic (NPY/AgRP) neurological signalling pathways. Interestingly, the reintroduction of chow following HFD had immediate and consistent responses between the sexes with body composition and most metabolic parameters normalised within 3 days. However, both sexes displayed elevated hypothalamic Npy levels reminiscent of starvation. The difference in RQ seen between the sexes on HFD was immediately abolished suggesting similar abilities to burn fat reserves for fuel.C57BL/6J mice have markedly different sex-specific behavioural and metabolic responses to the introduction as well as the sustained intake of a HFD, but consistent responses to a dieting situation.
TL;DR: This intervention's failure to impact body image and eating behaviors of college students illustrates the continuing challenge of eating disorders prevention.
TL;DR: What far‐reaching consequences dieting may produce in teenage girls is demonstrated and whether there is a direct relation between dieting and the development of anorexia nervosa is determined.
Abstract: The aim of the present study was to demonstrate what far-reaching consequences dieting may produce in teenage girls and to determine whether there is a direct relation between dieting and the development of anorexia nervosa. The study took the form of a longitudinal prospective investigation of dieting teenage schoolgirls over a 10-year observation period, particularly as regards physical and mental state of health and general psychosocial development.
The questionnaire study underlying the present work—entitled “The feeling of being fat and dieting in a school population”—was published in 1971 by Ingvar Nylander. With the aim of determining how often teenagers feel overweight, how often they diet and how often they develop disorders from dieting, Nylander interviewed 2370 students (1129 boys and 1241 girls). His study was published in Acta Socio-Medica Scand 1971, 1.
In the present work a primary material of 130 girls was selected from the dieting schoolgirls and 111 of these were examined by the author. The methods included a personal interview, examination of physical and mental health, an EEG examination and an intelligence test. Moreover, information was obtained concerning the subjects from their parents and from school health records and hospital case records. A follow-up study was done 10 years later, in which 97 of the subjects were again examined by the author while 12 subjects merely answered a questionnaire. The methods used were very similar to the foregoing ones. The subjects’dieting behaviour was evaluated on the basis of an anorectic behaviour scale. Further information was obtained from hospital case records.
The overall situation of the subjects was evaluated on the basis of case histories and of data from case records concerning heredity, environment, psychosocial adjustment, state of health and findings on examination.
Some of the subjects had had a dieting problem for a long time; they had generally dieted for short but repeated periods, but, in most cases, with little effect on their weight. Some patients had developed anorectic behaviour, or even AN, others did not. The former group had shown such behaviour at an early stage—or other signs of mental insufficiency—while the latter group was healthy and during dieting periods developed neither anorectic behaviour nor anorexia nervosa.
Thus no direct causal relation between dieting and anorexia nervosa could be demonstrated. If dieting is caused by mental insufficiency, this may lead to the development of anorectic behaviour and/or anorexia nervosa. However, it appears that dieting in a physically and mentally healthy teenage girl can hardly have such effects.
A prevalence evaluation of anorexia nervosa in this material showed higher values than those in previous studies.
TL;DR: Water and LCS beverage did not differ in their effects on total energy intake, macronutrient intakes or the selection of sweet foods and on motivational ratings in both LCS-naïve and LCS-habituated individuals.
TL;DR: In addition to anorexia nervosa and bulimia nervosa, DSM-IV designates a subclinical eating disorder category, eating disorder not otherwise specified (American Psychiatric Association, 1994).
Abstract: The criteria for a diagnosis of anorexia nervosa are summarized in Table 1. It is generally believed that fear of weight gain and body image disturbances are central features of anorexia nervosa that motivate extreme weight control methods such as fasting or purging (Williamson, 1990). The Diagnostic and Statistical Manual for Mental Disorders, 4th ed. (DSM-IV; American Psychiatric Association, 1994) designates two subtypes of anorexia nervosa. The restricting subtype describes anorexic patients who lose weight by restrictive eating and do not binge eat or purge. The binge eating/purging subtype describes anorexic patients who binge eat and/or purge. The DSM-IV criteria for a diagnosis of bulimia nervosa are summarized in Table 2. A central feature of bulimia nervosa is binge eating in which the individual uncontrollably eats a large amount of food in a short period of time. Compensatory behaviors such as self-induced vomiting, laxative and/or diuretic abuse, compulsive exercising, or fasting are presumed to be motivated by fear of weight gain and body image disturbances (Williamson, 1990). Two subtypes of bulimia nervosa are designated in DSM-IV. The purging subtype describes persons who binge eat, but use self-induced vomiting, laxative abuse, or diuretic abuse to prevent weight gain. The nonpurging subtype describes persons who binge eat, but use other forms of compensatory behavior such as dieting or excessive exercise to prevent weight gain. It is our experience that these patients are usually 10 to 15 pounds overweight. In addition to anorexia nervosa and bulimia nervosa, DSM-IV designates a subclinical eating disorder category, eating disorder not otherwise specified (American Psychiatric Association, 1994). Examples of this diagnosis include subthreshold anorexics who are not emaciated and obese binge eaters who binge eat but do not use vomiting or exercise to prevent weight gain (Williamson, Gleaves, & Savin, 1992).