Yolanda Alonso
Rovira i Virgili University
13 Papers
58 Citations
Yolanda Alonso is an academic researcher from Rovira i Virgili University. The author has contributed to research in topics: Medicine & Mitochondrial DNA. The author has an hindex of 7, co-authored 10 publications. Previous affiliations of Yolanda Alonso include Institut Pere Mata.
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Papers
Genetic and clinical evidence of mitochondrial dysfunction in autism spectrum disorder and intellectual disability
Alba Valiente-Pallejà,Helena Torrell,Gerard Muntané,Gerard Muntané,Maria José Cortés,Rafael Martínez-Leal,Nerea Abasolo,Yolanda Alonso,Elisabet Vilella,Lourdes Martorell +9 more
TL;DR: The high frequency of CAMDs, the low mtDNA content and the presence of putative pathogenic mtDNA mutations observed in both ASD and ID subjects are evidence of mitochondrial dysfunction in ASD andID.
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New evidence for the involvement of mitochondrial inheritance in schizophrenia: results from a cross-sectional study evaluating the risk of illness in relatives of schizophrenia patients.
Begoña Verge,Yolanda Alonso,Carmen Miralles,Joaquín Valero,Elisabet Vilella,Richard G. Boles,Lourdes Martorell +6 more
TL;DR: The results of this study support the hypothesis that mtDNA may be involved in schizophrenia, and in females, mtDNA could also beinvolved in the development of other psychiatric and nonpsychiatric conditions.
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Personality dimensions of schizophrenia patients compared to control subjects by gender and the relationship with illness severity
Carmen Miralles,Yolanda Alonso,Begoña Verge,Sònia Setó,Ana Milena Gaviria,L. Moreno,Maria José Cortés,Alfonso Gutiérrez-Zotes,Elisabet Vilella,Lourdes Martorell +9 more
TL;DR: InRevised dimensions were compared by gender between 161 schizophrenia patients and 214 healthy controls from a population-based sample using independent t-tests as mentioned in this paper. And they investigated whether personality dimensions are related to illness severity variables using correlation analyses and bivariate logistic regression, also by gender.
Increased blood lactate levels during exercise and mitochondrial DNA alterations converge on mitochondrial dysfunction in schizophrenia.
Alba Valiente-Pallejà,Helena Torrell,Yolanda Alonso,Elisabet Vilella,Gerard Muntané,Lourdes Martorell +5 more
TL;DR: These results are consistent with systemic mitochondrial malfunctioning in SZ and pinpoint lactate metabolism and mtDNA as targets for potential therapeutic treatments.
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Mitochondrial DNA (mtDNA) variants in the European haplogroups HV, JT, and U do not have a major role in schizophrenia.
Helena Torrell,Antonio Salas,Nerea Abasolo,Constanza Morén,Glòria Garrabou,Joaquín Valero,Yolanda Alonso,Elisabet Vilella,Javier Costas,Lourdes Martorell +9 more
TL;DR: The variants nominally associated with schizophrenia in the present study were not related either to phenotypic characteristics or to mitochondrial function, and evidence pointing to a role for mtDNA sequence variation in schizophrenia was found.
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