Yin Fu
Zhengzhou University
11 Papers
30 Citations
Yin Fu is an academic researcher from Zhengzhou University. The author has contributed to research in topics: Medicine & Biology. The author has an hindex of 2, co-authored 2 publications.
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Papers
MicroRNA-33a-5p suppresses esophageal squamous cell carcinoma progression via regulation of lncRNA DANCR and ZEB1.
TL;DR: It is suggested that the DANCR/miR-33a-5p/ZEB1 axis may be a potential prognostic and therapeutic target for microRNAs in the progression of esophageal squamous cell carcinoma.
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lncRNA MIAT promotes esophageal squamous cell carcinoma progression by regulating miR-1301-3p/INCENP axis and interacting with SOX2.
TL;DR: Findings indicated that MIAT promoted ESCC progression via targeting INCENP/miR‐1301‐3p axis and interacting with SOX2, suggesting novel potential therapeutic targets for ESCC.
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ATF3 in atherosclerosis: a controversial transcription factor
Bingyu Wang,Xi Yang,Xinyi Sun,Jianhui Liu,Yin Fu,Bing-Ya Liu,Jun Qiu,Jiangfang Lian,Jianqing Zhou +8 more
TL;DR: A new perspective for Atherosclerosis therapy is provided by summarizing the mechanism of ATF3 in atherosclerosis, as well as the structure and pathophysiological properties of ATF1 and ATF3.
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4-phenylbutyric acid re-trafficking hERG/G572R channel protein by modulating the endoplasmic reticulum stress-associated chaperones and endoplasmic reticulum-associated degradation gene.
Wen-Chien Tang,Dihui Cai,Yin Fu,Zequn Zheng,Xiaoyan Huang,Rami N. Khouzam,Yongfei Song,Jiangfang Lian +7 more
TL;DR: 4-PBA corrects hERG channel transport defects by inhibiting excessive ERS and the endoplasmic reticulum-associated degradation (ERAD)-related gene E3 ubiquitin ligase HRD1, and improved WT/G572R channel current.
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The Advantages, Challenges, and Future of Human-Induced Pluripotent Stem Cell Lines in Type 2 Long QT Syndrome
Dihui Cai,Zequn Zheng,Xiaojun Jin,Yin Fu,Lichao Cen,Jiachun Ye,Yongfei Song,Jiangfang Lian +7 more
TL;DR: This review discusses how hiPSC-CM and gene editing are used to decipher mechanisms of LQT2, screen for cardiotoxicity, and identify therapeutic strategies, thus promoting the realization of precision medicine for L QT2 patients.
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