Ye Tian
Jilin University
11 Papers
Ye Tian is an academic researcher from Jilin University. The author has contributed to research in topics: AMPK & Protein kinase A. The author has an hindex of 9, co-authored 10 publications.
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Papers
Corilagin alleviates acetaminophen-induced hepatotoxicity via enhancing the AMPK/GSK3β-Nrf2 signaling pathway
TL;DR: In vivo, Cori significantly protected against APAP-induced ALF by reducing mortality and alanine transaminase (ALT) and aspartate aminotransferase (AST) levels, attenuating histopathological liver changes, and increasing the superoxide dismutase (SOD) content and GSH-to-GSSG ratio as well as suppressing c-jun N-terminal kinase (JNK) phosphorylation.
Protective effects of morin on lipopolysaccharide/d-galactosamine-induced acute liver injury by inhibiting TLR4/NF-κB and activating Nrf2/HO-1 signaling pathways.
TL;DR: The results suggested that morin could exert the anti‐inflammatory and anti‐oxidative effects against LPS/D‐GalN‐induced acute liver injury by activating Nrf2 signal pathways and inhibiting NF‐&kgr;B activation.
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Isorhamnetin ameliorates LPS-induced inflammatory response through downregulation of NF-κB signaling
TL;DR: Isorhamnetin might be a potential therapeutic agent for preventing inflammatory diseases by suppressing NF-κB signaling and reducing the amount of inflammatory cells, the lung wet-to-dry weight ratio, protein leakage, and myeloperoxidase activity.
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Rosmarinic acid protects mice from lipopolysaccharide/d-galactosamine-induced acute liver injury by inhibiting MAPKs/NF-κB and activating Nrf2/HO-1 signaling pathways.
Zheng Li,Haihua Feng,Yue Wang,Bingyu Shen,Ye Tian,Lin Wu,Qiaoling Zhang,Meiyu Jin,Guowen Liu +8 more
TL;DR: RA could protect C57BL/6 mice from LPS/d-GalN-induced acute liver injury, which showed that RA could enhance the level of glutathione-dependent peroxidase (GSH-PX) and promote that nuclear factor erythroid-2-related factor 2 (Nrf2) transported into nucleus.
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Magnolol Alleviates Inflammatory Responses and Lipid Accumulation by AMP-Activated Protein Kinase-Dependent Peroxisome Proliferator-Activated Receptor α Activation.
Ye Tian,Haihua Feng,Lu Han,Lin Wu,Hongming Lv,Bingyu Shen,Zheng Li,Qiaoling Zhang,Guowen Liu +8 more
TL;DR: Results suggest that MG exerts protective effects against steatosis, hyperlipidemia, and the underlying mechanism, which may be closely associated with AKT/AMPK/PPARα activation and MAPK/NF-κB/SREBP-1c inhibition.