Wei Zhang
Kanazawa Medical University
22 Papers
93 Citations
Wei Zhang is an academic researcher from Kanazawa Medical University. The author has contributed to research in topics: Portal venous pressure & Central venous pressure. The author has an hindex of 9, co-authored 22 publications.
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Papers
Pulmonary vasoconstrictive and bronchoconstrictive responses to anaphylaxis are weakened via β2-adrenoceptor activation by endogenous epinephrine in anesthetized rats.
TL;DR: The pulmonary vasoconstrictive and bronchoconStrictive responses to systemic anaphylaxis were weakened via &bgr;2-adrenoceptor activation by epinephrine endogenously released from the adrenal gland in the anesthetized Sprague-Dawley rats.
Involvement of splanchnic vascular bed in anaphylactic hypotension in anesthetized BALB/c mice
Wei Liu,Hiromichi Takano,Toshishige Shibamoto,Sen Cui,Zhansheng Zhao,Wei Zhang,Yasutaka Kurata +6 more
TL;DR: Results suggest that splanchnic vascular beds are involved in BALB/c mouse anaphylactic hypotension, and presumably act as sources of chemical mediators to cause theAnaphylaxis-induced portal hypertension, resulting in a decrease in circulating blood volume and, thus, systemic arterial hypotension.
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Venous resistance increases during rat anaphylactic shock.
TL;DR: In rat anaphylactic shock, a substantial increase in Rv presumably due to hepatic venoconstriction may decrease venous return, resulting in systemic hypotension.
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•Journal Article
Rat hepatic and splanchnic vascular responses to anaphylactic shock, compared with hemorrhagic or vasodilator-induced shock.
Wei Zhang,Toshishige Shibamoto,Mamoru Tanida,Mofei Wang,Mofei Wang,Lingling Sun,Lingling Sun,Yasutaka Kurata +7 more
TL;DR: Hepatic and splanchnic vascular responses differ according to the type of shock, and anaphylactic hypotension is characterized by markedly increased portal venous resistance.
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NG-Nitro- L -arginine Methyl Ester, but Not Methylene Blue, Attenuates Anaphylactic Hypotension in Anesthetized Mice
TL;DR: It is shown in anesthetized BALB/c mice that L-NAME attenuated anaphylactic hypotension without affecting portal hypertension, and this beneficial effect appears not to depend on the soluble guanylate cyclase pathway.
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