Tao Lin
Chongqing Medical University
60 Papers
106 Citations
Tao Lin is an academic researcher from Chongqing Medical University. The author has contributed to research in topics: Medicine & Sertoli cell. The author has an hindex of 18, co-authored 59 publications.
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Papers
Dynamic Effect of Di-2-(Ethylhexyl) Phthalate on Testicular Toxicity: Epigenetic Changes and Their Impact on Gene Expression
TL;DR: Changes in DNA methylation may play an important role in abnormal testicular function caused by environmental factors such as maternal exposure to DEHP, which may be a mechanism of DEHP-mediated testicular toxicity.
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Potential Spermatogenesis Recovery with Bone Marrow Mesenchymal Stem Cells in an Azoospermic Rat Model
Deying Zhang,Xing Liu,Jinpu Peng,Dawei He,Tao Lin,Jing Zhu,Xuliang Li,Yuanyuan Zhang,Guanghui Wei +8 more
TL;DR: The present study indicates that BMSCs might offer alternative treatment for the patients with azoospermatic infertility after cancer chemotherapy, by providing the potential to trans-differentiate into spermatogenic-like-cells, enhancing endogenous fertility recovery.
Urethral reconstruction with autologous urine-derived stem cells seeded in three-dimensional porous small intestinal submucosa in a rabbit model
Yang Liu,Yang Liu,Wenjun Ma,Wenjun Ma,Bo Liu,Bo Liu,Yangcai Wang,Yangcai Wang,Jiaqiang Chu,Geng Xiong,Geng Xiong,Lianju Shen,Chunlan Long,Tao Lin,Dawei He,Denis Butnaru,Lyundup Alexey,Yuanyuan Zhang,Deying Zhang,Deying Zhang,Guanghui Wei +20 more
TL;DR: The feasibility of using autologous urine-derived stem cells (USCs) seeded on small intestinal submucosa (SIS) to repair a urethral defect in a rabbit model and demonstrating that the transplanted USCs differentiated into UCs and SMCs in vivo is demonstrated.
Spermatogenesis dysfunction induced by PM2.5 from automobile exhaust via the ROS-mediated MAPK signaling pathway.
Bin Liu,Shengde Wu,Lianju Shen,Tian-Xin Zhao,Yi Wei,Xiangliang Tang,Chunlan Long,Yue Zhou,Dawei He,Tao Lin,Guanghui Wei +10 more
TL;DR: The results suggest that PM2.5 from automobile exhaust disrupted spermatogenesis via ROS-mediated MAPK pathways, and that a combined vitamin C and E intervention effectively mitigated toxicity in the male reproductive system.
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Automobile exhaust-derived PM2.5 induces blood-testis barrier damage through ROS-MAPK-Nrf2 pathway in sertoli cells of rats.
Bin Liu,Lianju Shen,Tian-Xin Zhao,Mang Sun,Junke Wang,Chunlan Long,Dawei He,Tao Lin,Shengde Wu,Guanghui Wei +9 more
TL;DR: Findings indicate that PM2.5 derived from automobile exhaust causes oxidative stress, which in turn causes cellular apoptosis of SCs and damage of the blood-testis barrier, resulting male spermatogenesis dysfunction, in which ROS-MAPK-Nrf-2 pathways may play a key role.
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