Shanglang Cai
Qingdao University
8 Papers
21 Citations
Shanglang Cai is an academic researcher from Qingdao University. The author has contributed to research in topics: Catheter ablation & Cardioversion. The author has an hindex of 4, co-authored 8 publications.
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Papers
Ablation alleviates atrial fibrillation by regulating the signaling pathways of endothelial nitric oxide synthase/nitric oxide via miR-155-5p and miR-24-3p.
TL;DR: This study aimed to identify the microRNAs implicated in atrial fibrillation to investigate the molecular mechanisms underlying the role of micro RNAs in ablation‐based AF treatment.
18
Comparison of Direct Current Synchronized Cardioversion to Ibutilide-Guided Catheter Ablation for Long-Term Sinus Rhythm Maintenance After Isolated Pulmonary Vein Isolation of Persistent Atrial Fibrillation
TL;DR: In persistent AF treatment, ibutilide-guided ablation after PVI yields higher 1-year SR maintenance rate than PVI only, which is significantly higher than before or after reablation.
7
Patent
Hemostasis device for department of cardiology
Maojing Wang,Yang Ji,Wei Ding,Shanglang Cai +3 more
- 30 Mar 2016
TL;DR: In this paper, a hemostasis device for department of cardiology is described, which consists of a main box body, supporting legs fixedly arranged on the bottom face inside the main box, the top ends of the supporting legs are provided with a supporting plate, the upper surface of the plate is fixedly provided with self-locking nut, and the selflocking nut is connected with a lead screw shaft through a thread pair in a matched mode.
4
Efficacy and effects on cardiac function of radiofrequency catheter ablation vs. direct current cardioversion of persistent atrial fibrillation with left ventricular systolic dysfunction.
TL;DR: Compared with non-responders, patients with improved LVEF post-ablation had poorer ventricular rate control and cardiac function at baseline, suggesting a significant component of tachycardia-induced cardiomyopathy in this group.
Co-administration of Colchicine and Allopurinol alleviates vascular calcification induced by hyperuricemia via suppressing inflammation and oxidative stress
TL;DR: This work gained a deeper insight into the molecular mechanism of hyperuricemia, and revealed that the efficiency of the combined treatment with COL and ALL out-performed the mono-therapy of any single compound.