Randong Yang
Wenzhou Medical College
9 Papers
1 Citations
Randong Yang is an academic researcher from Wenzhou Medical College. The author has contributed to research in topics: Medicine & Biology. The author has an hindex of 1, co-authored 2 publications.
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Papers
Suppression of endoplasmic reticulum stress-dependent autophagy enhances cynaropicrin-induced apoptosis via attenuation of the P62/Keap1/Nrf2 pathways in neuroblastoma
Randong Yang,Shu-Yan Ma,Ran Zhuo,Lingqi Xu,Si-Qi Jia,Pengcheng Yang,Ye Yao,Hai-bo Cao,Liya Ma,Jian Pan,Jin Wang +10 more
TL;DR: It is suggested that CYN may be a promising candidate for the treatment of NB, and the combination of pharmacological inhibitors of autophagy may hold novel therapeutic potential for the Treatment of NB.
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Super-enhancer-associated INSM2 regulates lipid metabolism by modulating mTOR signaling pathway in neuroblastoma
Hai-bo Cao,Ran Zhuo,Zimu Zhang,Jianwei Wang,Yan-Fang Tao,Randong Yang,Xinyi Guo,Yanling Chen,Si-Qi Jia,Yebin Yao,Pengcheng Yang,Juan-juan Yu,Wan Jiao,Xiaolu Li,Fang Fang,Yi Xie,Gen Li,Di Wu,Hai-Rong Wang,Chenxi Feng,Yunyun Xu,Zhiheng Liu,Jia Hong Pan,Jian Wang +23 more
TL;DR: In this article , the insulinoma-associated 2 gene (INSM2) was found to be improperly controlled by super enhancers, a mammalian genome region that has been shown to control the expression of NB cell identity genes.
Super-enhancer-associated TTC8 alters the nucleocytoplasmic distribution of PHOX2B and activates MAPK signaling in neuroblastoma
Ran Zhuo,Zimu Zhang,Xinyi Guo,Hai-bo Cao,Yanling Chen,Yan-Fang Tao,Randong Yang,Xiaolu Li,Jian Pan,Jin Wang +9 more
TL;DR: TTC8, a member of the BBS family of proteins involved in ciliary transport, is the authors' newly identified super-enhancer-associated gene in NB, and its roles in tumorigenesis remain elusive.
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Pediatric Patients with Adrenal Neuroblastoma: A SEER Analysis, 2004–2013:
TL;DR: Pediatric patients with adrenal NB have significantly worse survival than those with nonadrenal NB, and there was no survival difference for patients to receive CR and no CR.
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Novel pharmacological inhibition of JMJD3 improves necrotizing enterocolitis by attenuating the inflammatory response and ameliorating intestinal injury.
Shu-Yan Ma,Lingqiu Xu,Lulu Chen,Xu Sun,Fang-lin Hu,Yuan Gong,Randong Yang,Jing Li,Qian Wang,Shungen Huang,Huiting Zhou,Jian Wang +11 more
TL;DR: In this article , the authors showed that upregulated JMJD3 and suppressed histone H3 lysine 27 (H3K27) demethylase plays a proinflammatory role in sepsis and neuroinflammation.