Peter H. Lin
Veterans Health Administration
57 Papers
575 Citations
Peter H. Lin is an academic researcher from Veterans Health Administration. The author has contributed to research in topics: Angioplasty & Aneurysm. The author has an hindex of 25, co-authored 56 publications. Previous affiliations of Peter H. Lin include Emory University Hospital & Michael E. DeBakey Veterans Affairs Medical Center in Houston.
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Papers
Chemical and molecular mechanisms of antioxidants: experimental approaches and model systems.
TL;DR: Inhibition of free radical generating enzymes is inhibition of internal antioxidant enzymes and prevention of lipid peroxidation is prevention of DNA damage and protein modification.
1.2K
Outcomes Following Endovascular vs Open Repair of Abdominal Aortic Aneurysm: A Randomized Trial
Frank A. Lederle,Julie A. Freischlag,Tassos C. Kyriakides,Frank T. Padberg,Jon S. Matsumura,Ted R. Kohler,Peter H. Lin,Jessie M. Jean-Claude,Dolores F. Cikrit,Kathleen M. Swanson,Peter Peduzzi +10 more
TL;DR: Perioperative mortality was low for both procedures and lower for endovascular than open repair in this report of short-term outcomes after elective AAA repair, and the early advantage of endov vascular repair was not offset by increased morbidity or mortality in the first 2 years after repair.
1.1K
Resistin decreases expression of endothelial nitric oxide synthase through oxidative stress in human coronary artery endothelial cells
Changyi Chen,Changyi Chen,Jun Jiang,Jian-Ming Lü,Hong Chai,Xinwen Wang,Peter H. Lin,Peter H. Lin,Qizhi Yao,Qizhi Yao +9 more
TL;DR: Resistin directly induces eNOS downregulation through overproduction of ROS and activation of p38 and JNK in HCAECs, which indicates Resistin-induced mitochondrial dysfunction and imbalance in cellular redox enzymes may be the underlying mechanisms of oxidative stress.
197
Endoluminal stent placement and coil embolization for the management of carotid artery pseudoaneurysms
TL;DR: Endovascular treatment of carotid artery pseudoaneurysms is a useful alternative to standard surgical repair, and avoids the necessity for surgical exposure at the skull base with its inherent morbidity.
127
Shear stress regulates occludin and VEGF expression in porcine arterial endothelial cells.
Brian S. Conklin,Dian-sheng Zhong,Dian-sheng Zhong,Weidong Zhao,Weidong Zhao,Peter H. Lin,Peter H. Lin,Changyi Chen,Changyi Chen +8 more
TL;DR: Investigation of the effects of shear stress on the expression of occludin and vascular endothelial growth factor and VEGF shows changes that may suggest a possible molecular mechanism for increased endothelial permeability due to lowShear stress.
103