Juan Antonio Mico
University of Cádiz
144 Papers
1K Citations
Juan Antonio Mico is an academic researcher from University of Cádiz. The author has contributed to research in topics: Chronic pain & Neuropathic pain. The author has an hindex of 39, co-authored 143 publications. Previous affiliations of Juan Antonio Mico include University of the Basque Country & Instituto de Biologia Molecular e Celular.
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Papers
A review of chronic pain impact on patients, their social environment and the health care system.
TL;DR: It is concluded that in addition to the serious consequences on the patient’s life, CP has a severe detrimental effect on their social and family environment, as well as on health care services.
Antidepressants and pain
TL;DR: In this paper, Tricyclic antidepressants are considered to be first-line drugs for the treatment of neuropathic pain, and they are shown to be analgesic in patients with chronic pain and no concomitant depression.
397
Noradrenergic Locus Coeruleus pathways in pain modulation.
Meritxell Llorca-Torralba,Gisela Borges,Fani Neto,Juan Antonio Mico,Esther Berrocoso,Esther Berrocoso +5 more
TL;DR: The main goal of this review is to bring together the data available regarding the noradrenergic system in relation to pain, particularly focusing on the ascending and descending LC projections in different conditions.
200
Pro-/Anti-inflammatory Dysregulation in Patients With First Episode of Psychosis: Toward an Integrative Inflammatory Hypothesis of Schizophrenia
Borja García-Bueno,Miquel Bioque,Karina S. MacDowell,M. Fe Barcones,Mónica Martínez-Cengotitabengoa,Laura Pina-Camacho,Roberto Rodriguez-Jimenez,Pilar A. Saiz,Carmen Castro,Amalia Lafuente,Javier Santabárbara,Ana González-Pinto,Mara Parellada,Gabriel Rubio,M. Paz García-Portilla,Juan Antonio Mico,Miguel Bernardo,Juan C. Leza +17 more
TL;DR: The results of this study indicate robust phenotypical differences at the cellular machinery level in PMBC of patients with FEP, suggesting the anti-inflammatory mediator 15d-PGJ2 might be used as plasmatic biomarker for first episodes of psychosis.
Origin and consequences of brain Toll-like receptor 4 pathway stimulation in an experimental model of depression
Iciar Gárate,Borja García-Bueno,José L. M. Madrigal,Lidia Bravo,Esther Berrocoso,Javier R. Caso,Juan Antonio Mico,Juan C. Leza +7 more
TL;DR: The results suggest that LPS from bacterial translocation is responsible, at least in part, for the TLR-4 activation found in brain after CMS, which leads to release of inflammatory mediators in the CNS.