5 Papers
85 Citations
Jia Su is an academic researcher from Beijing University of Chemical Technology. The author has contributed to research in topics: Phosphorylation & Inflammation. The author has an hindex of 5, co-authored 5 publications.
Chat about Author
Papers
The G-Protein-Coupled Bile Acid Receptor Gpbar1 (TGR5) Inhibits Gastric Inflammation Through Antagonizing NF-κB Signaling Pathway.
Cong Guo,Hui Qi,Yingjie Yu,Qiqi Zhang,Jia Su,Donna Yu,Wendong Huang,Wei-Dong Chen,Wei-Dong Chen,Yan-Dong Wang +9 more
TL;DR: TGR5 activation antagonized NF-κB signaling pathway through suppressing its transcription activity, the phosphorylation of IκBα and p65 translocation, which suggests that TGR5 antagonizes gastric inflammation at least in part by inhibiting NF-kkB signaling.
The G-protein-coupled bile acid receptor Gpbar1 (TGR5) suppresses gastric cancer cell proliferation and migration through antagonizing STAT3 signaling pathway
Cong Guo,Jia Su,Zhijun Li,Rui Xiao,Jianxun Wen,Yanyan Li,Meng Zhang,Xueting Zhang,Donna Yu,Wendong Huang,Wei-Dong Chen,Wei-Dong Chen,Yan-Dong Wang +12 more
TL;DR: Findings suggest that TGR5 antagonizes gastric cancer proliferation and migration at least in part by inhibiting STAT3 signaling, which may serve as an attractive therapeutic tool for human gastric cancers.
The G-protein-coupled bile acid receptor Gpbar1 (TGR5) protects against renal inflammation and renal cancer cell proliferation and migration through antagonizing NF-κB and STAT3 signaling pathways.
Jia Su,Qiqi Zhang,Hui Qi,Linlin Wu,Yuanqiang Li,Donna Yu,Wendong Huang,Wei-Dong Chen,Wei-Dong Chen,Yan-Dong Wang +9 more
TL;DR: The results suggest that TGR5 antagonizes kidney inflammation and kidney cancer cell proliferation and migration at least in part by inhibiting NF-κB and STAT3 signaling.
MicroRNA-149* suppresses hepatic inflammatory response through antagonizing STAT3 signaling pathway
Qiqi Zhang,Jia Su,Ziwei Wang,Hui Qi,Zeyong Ge,Zhijun Li,Wei-Dong Chen,Wei-Dong Chen,Yan-Dong Wang +8 more
TL;DR: Findings identify miR-149* as a negative mediator of inflammation that may serve as an attractive therapeutic tool for immune and inflammatory liver diseases.
Quercetin Inhibits LPS-Induced Inflammation and ox-LDL-Induced Lipid Deposition.
Feng Xue,Xiaobo Nie,Jianping Shi,Qingxue Liu,Ziwei Wang,Xiting Li,Jinqiu Zhou,Jia Su,Mingming Xue,Wei-Dong Chen,Wei-Dong Chen,Yan-Dong Wang +11 more
TL;DR: The data reveal that QCT has obvious anti-inflammatory and antioxidant virtues and could be a therapeutic agent for the prevention and treatment of AS.