Graham Jenkin
Hudson Institute of Medical Research
214 Papers
1.6K Citations
Graham Jenkin is an academic researcher from Hudson Institute of Medical Research. The author has contributed to research in topics: Medicine & Fetus. The author has an hindex of 42, co-authored 201 publications. Previous affiliations of Graham Jenkin include Hudson Institute & Monash Institute of Medical Research.
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Papers
Neonatal Morbidities of Fetal Growth Restriction: Pathophysiology and Impact.
Atul Malhotra,Beth J. Allison,Margie Castillo-Melendez,Margie Castillo-Melendez,Graham Jenkin,Graham Jenkin,Graeme R. Polglase,Graeme R. Polglase,Suzanne L. Miller,Suzanne L. Miller +9 more
TL;DR: A better understanding of neonatal morbidities associated with FGR will enable early neonatal detection, monitoring and management of potential adverse outcomes in the newborn period and beyond.
A Review of Animal Models of Intervertebral Disc Degeneration: Pathophysiology, Regeneration, and Translation to the Clinic
TL;DR: The role of animal models of disc degeneration in translational research leading to clinical trials of novel cellular therapies is explored and the purpose of such animal models is to further understanding and, ultimately, improve treatment of Disc degeneration.
Amnion epithelial cell isolation and characterization for clinical use.
Sean V. Murphy,Sharina Rosli,Rutu Acharya,Louisa Mathias,Rebecca Lim,Euan M. Wallace,Graham Jenkin +6 more
TL;DR: The differentiation potential of hAECs isolated using this animal product-free method was demonstrated by differentiation into lineages of the three primary germ layers and expression of lineage-specific markers analyzed by PCR, immunocytochemistry, and histology.
165
Human Amnion Epithelial Cells Prevent Bleomycin-Induced Lung Injury and Preserve Lung Function:
Sean V. Murphy,Rebecca Lim,Hayley Dickinson,Rutu Acharya,Sharina Rosli,Graham Jenkin,Euan M. Wallace,Euan M. Wallace +7 more
TL;DR: It is demonstrated that, compared to bleomycin alone, IP administration of hAECs 24 h after bleomcyin, decreased gene expression of the proinflammatory cytokines TNF-α, TGF-β, IFN-γ, and IL-6 and decreased subsequent pulmonary fibrosis with less pulmonary collagen deposition, reduced levels of α-smooth muscle actin and decreased inflammatory cell infiltrate.
153
Melatonin provides neuroprotection in the late-gestation fetal sheep brain in response to umbilical cord occlusion
TL;DR: Brief asphyxia results in significant and delayed entry of ·OH into the extracellular space of cortical gray matter in the fetal sheep brain, and melatonin given to the mother at the time of the insult abrogates this increase.
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