Giuseppina Di Stefano
Nuclear Regulatory Commission
63 Papers
674 Citations
Giuseppina Di Stefano is an academic researcher from Nuclear Regulatory Commission. The author has contributed to research in topics: Mitochondrion & Dentate gyrus. The author has an hindex of 22, co-authored 61 publications. Previous affiliations of Giuseppina Di Stefano include University of Camerino.
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Papers
One-month strawberry-rich anthocyanin supplementation ameliorates cardiovascular risk, oxidative stress markers and platelet activation in humans.
José M. Alvarez-Suarez,Francesca Giampieri,Sara Tulipani,Tiziana Casoli,Giuseppina Di Stefano,Ana M. González-Paramás,Celestino Santos-Buelga,Franco Busco,José L. Quiles,Mario D. Cordero,Stefano Bompadre,Bruno Mezzetti,Maurizio Battino +12 more
TL;DR: Strawberries consumption improves plasma lipids profile, biomarkers of antioxidant status, antihemolytic defenses and platelet function in healthy subjects, encouraging further evaluation on a population with higher cardiovascular disease risk.
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Dose-dependent opposite effect of zinc on apoptosis in mouse thymocytes.
TL;DR: Evaluated data show that in vitro Zn2+ has a dose-dependent opposite effect on apoptosis, suggesting that Zn 2+ not only acts as an inhibitor but also plays a more complex role in physiological intrathymic cell selection.
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Effect of melatonin and pineal grafting on thymocyte apoptosis in aging mice.
TL;DR: The results suggest that MEL treatment or PG prevent age-related thymus involution through regulation of thymocyte apoptosis which, in turn, occurs through modulation of the pituitary-adrenal axis and zinc turnover determined by the pineal hormone.
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Optimization of cytotoxic assay by target cell retention of the fluorescent dye carboxyfluorescein diacetate (CFDA) and comparison with conventional 51CR release assay.
TL;DR: Comparison between a fluorimetric NK assay based on the target cell retention of fluorescent dye carboxyfluorescein diacetate (cFDA) and standard 51Cr release assay shows that the measurements of cFDA retained by target cells represent a method of evaluating cytotoxicity completely comparable to the 51 Cr release assay.
64
Release of beta-amyloid from high-density platelets: implications for Alzheimer's disease pathology.
Tiziana Casoli,Giuseppina Di Stefano,Belinda Giorgetti,Yessica Grossi,Marta Balietti,Patrizia Fattoretti,Carlo Bertoni-Freddari +6 more
TL;DR: The data suggest that high-density platelets undergo activation likely by increased frequency of platelet-platelet collisions, which determines the activation of APP beta-processing with consequent release of Abeta.
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