Fengjuan Wang
University College Dublin
10 Papers
Fengjuan Wang is an academic researcher from University College Dublin. The author has contributed to research in topics: Medicine & Biology. The author has an hindex of 4, co-authored 4 publications. Previous affiliations of Fengjuan Wang include University of Strasbourg & Spanish National Research Council.
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Papers
The biomolecular corona is retained during nanoparticle uptake and protects the cells from the damage induced by cationic nanoparticles until degraded in the lysosomes.
Fengjuan Wang,Fengjuan Wang,Lu Yu,Marco P. Monopoli,Peter Sandin,Eugene Mahon,Anna Salvati,Kenneth A. Dawson +7 more
TL;DR: This study investigates the effects of protein corona that normally forms on the surface of nanoparticles during in vivo use, describing the steps of intracellular processing of such particles, to enhance the understanding of how these particles interact with the cellular machinery.
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Lysosome-dependent cell death and deregulated autophagy induced by amine-modified polystyrene nanoparticles
TL;DR: Though the cells initially seem to induce autophagy as a surviving mechanism, the damage of NH2-PS NPs to lysosomes probably results in lysOSomal dysfunctions, leading to blockage of autophagic flux at the level of lYSosomes and the eventual cell death.
Inhibiting specificity protein 1 attenuated sevoflurane-induced mitochondrial stress and promoted autophagy in hippocampal neurons through PI3K/Akt/mTOR and α7-nAChR signaling
TL;DR: In this paper , the role of specificity protein 1 (SP1) in mediating mitochondrial stress and autophagy in neurons under sevoflurane exposure was investigated.
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ESM-1 Mediates Cell Progression in Clear Cell Renal Cell Carcinoma by Affecting Wnt/β-Catenin Signalling Pathway.
TL;DR: The ESM-1 expression increased in ccRCC tissue samples and cells, which indicated poor prognosis as mentioned in this paper , and the ESM1 silencing considerably inhibited CCRCC cell growth, invasion and migration in vitro.
3
USP18 alleviates neurotoxicity induced by sevoflurane via AKT and NF-κB pathways
TL;DR: Mechanically, USP18 reduced sevoflurane-induced neurotoxicity via AKT and NF-κB pathway and attenuated sev oflurain-induced neuronal apoptosis.