David E. Shaw
University of Birmingham
9 Papers
55 Citations
David E. Shaw is an academic researcher from University of Birmingham. The author has contributed to research in topics: Retinopathy of prematurity & Medicine. The author has an hindex of 7, co-authored 9 publications. Previous affiliations of David E. Shaw include University of Leicester.
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Papers
Randomised, Placebo‐Controlled Study of Vigabatrin as First‐Line Treatment of Infantile Spasms
TL;DR: This prospective, randomised, and placebo‐controlled trial of VGB in infantile spasms was considered to be justified and feasible to confirm or refute these previous findings.
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Natural history of retinopathy of prematurity: a prospective study.
Alistair R. Fielder,Alistair R. Fielder,David E. Shaw,David E. Shaw,Judith Robinson,Judith Robinson,Yin K Ng,Yin K Ng +7 more
TL;DR: The natural history of retinopathy of prematurity has been studied prospectively using a protocol designed specifically to investigate the subtle features of this condition and the relevance of these findings to ROP screening is discussed.
Retinopathy of prematurity: a prospective study. Review at six months.
D Laws,David E. Shaw,David E. Shaw,Judith Robinson,Judith Robinson,Helen S Jones,Y K Ng,Alistair R. Fielder,Alistair R. Fielder +8 more
TL;DR: A cohort of infants of birthweight andleq;s 1700 g studied prospectively for retinopathy of prematurity (ROP) has been reviewed at 6 months corrected age and the findings related to the neonatal data, with a significant trend towards myopia.
Time-related Variation in Normal Automated Static Perimetry
TL;DR: Both global and pointwise group mean sensitivity decreased in a time-related manner, deterioration being greater for the second eye at each visit, for both 200 ms and 100 ms stimulus durations.
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Light and retinopathy of prematurity: does retinal location offer a clue?
TL;DR: Radiometric and physiological evidence suggests that the very immature neonate, most at risk of developing severe ROP, receives the greatest retinal irradiance, and ROP commences in the areas of the retina receiving the highest light dose, and its onset is either retarded or inhibited in the darker retinal regions.
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