Daniel P. Holschneider
University of Southern California
108 Papers
1.1K Citations
Daniel P. Holschneider is an academic researcher from University of Southern California. The author has contributed to research in topics: Brain mapping & Cardiac output. The author has an hindex of 29, co-authored 104 publications. Previous affiliations of Daniel P. Holschneider include Veterans Health Administration & Semel Institute for Neuroscience and Human Behavior.
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Papers
A gut-derived metabolite alters brain activity and anxiety behaviour in mice
Brittany D. Needham,Masanori Funabashi,Mark D. Adame,Zhuo Wang,Joseph C. Boktor,Jillian R. Haney,Weili Wu,Claire Rabut,Mark S. Ladinsky,Son-Jong Hwang,Yumei Guo,Qiyun Zhu,Jessica A. Griffiths,Rob Knight,Pamela J. Bjorkman,Mikhail G. Shapiro,Daniel H. Geschwind,Daniel P. Holschneider,Michael A. Fischbach,Sarkis K. Mazmanian +19 more
TL;DR: In this paper , the authors identified biosynthetic genes from the gut microbiome that mediate the conversion of dietary tyrosine to 4-ethylphenol (4EP), and bioengineered gut bacteria to selectively produce 4EP in mice.
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A spontaneous point mutation produces monoamine oxidase A/B knock-out mice with greatly elevated monoamines and anxiety-like behavior.
TL;DR: Observed chase/escape and anxiety-like behavior in theMAO A/B KO mice, different from MAO A or B singleKO mice, suggest that varying monoamine levels result in both a unique biochemical and behavioral phenotype.
146
The Effects of Exercise on Dopamine Neurotransmission in Parkinson's Disease: Targeting Neuroplasticity to Modulate Basal Ganglia Circuitry.
Giselle M. Petzinger,Daniel P. Holschneider,Beth E. Fisher,Sarah McEwen,Natalie Kintz,Matthew R. Halliday,William A. Toy,J.W. Walsh,Jeff A. Beeler,Michael W. Jakowec +9 more
- 01 Jan 2015
TL;DR: It is hypothesize that the potential effects of exercise on inducing neuroplasticity in a circuit specific manner may occur through synergistic mechanisms that include the coupling of an increasing neuronal metabolic demand and increased blood flow.
Memory, Mood, Dopamine, and Serotonin in the 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine-Lesioned Mouse Model of Basal Ganglia Injury
Marta G. Vučković,Ruth I. Wood,Daniel P. Holschneider,Avery Abernathy,Daniel M. Togasaki,Alexsandra Smith,Giselle M. Petzinger,Michael W. Jakowec +7 more
TL;DR: In conclusion, acute MPTP lesioning regimen in mice causes impairments in associative memory and conditioned fear, no mood changes, and depletion of dopamine and serotonin throughout the brain.
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Reorganization of functional brain maps after exercise training: Importance of cerebellar-thalamic-cortical pathway.
TL;DR: Following ET, the new dynamic equilibrium of the brain is characterized by increases in the efficiency of neural processing and an increased influence of the CbTC circuit, and Cerebral regions demonstrating changes in neural activation may point to alternate circuits, which may be mobilized during neurorehabilitation.
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