Daniel Alves Rosa
Universidade Federal de Goiás
21 Papers
163 Citations
Daniel Alves Rosa is an academic researcher from Universidade Federal de Goiás. The author has contributed to research in topics: Hypertonic saline & Median preoptic nucleus. The author has an hindex of 8, co-authored 19 publications. Previous affiliations of Daniel Alves Rosa include Federal University of São Paulo.
Chat about Author
Papers
A2 Noradrenergic Lesions Prevent Renal Sympathoinhibition Induced by Hypernatremia in Rats
Gustavo Rodrigues Pedrino,André Henrique Freiria-Oliveira,Debora S. A. Colombari,Daniel Alves Rosa,Sergio L. Cravo +4 more
TL;DR: The results suggest that A2 noradrenergic neurons are components of the neural pathways regulating the composition of the extracellular fluid compartment and are selectively involved in hypernatremia-induced sympathoinhibition.
Renal sympathoinhibition induced by hypernatremia: Involvement of A1 noradrenergic neurons
TL;DR: The results suggest that A1 noradrenergic neurons are involved in the sympathoinhibition and consequent renal vasodilatation to acute changes in the extracellular fluid compartment.
31
Dependência da prática de exercícios físicos: estudo com maratonistas brasileiros
TL;DR: The mean score in the Brazilian sample was sim-ilar to that described by the authors of the original instru-ment, suggesting that the translation did not affect the sensitivity of the instrument and that it can be useful in the study of running or physical activity dependence in Brazil-ian athletes.
A1 noradrenergic neurons lesions reduce natriuresis and hypertensive responses to hypernatremia in rats.
Elaine da Silva,André Henrique Freiria-Oliveira,Carlos Henrique Xavier Custódio,Paulo César Ghedini,Luiz Artur Mendes Bataus,Eduardo Colombari,Carlos Henrique de Castro,Diego Basile Colugnati,Daniel Alves Rosa,Sergio L. Cravo,Gustavo Rodrigues Pedrino +10 more
TL;DR: It is suggested that medullary noradrenergic A1 neurons are involved in the excitatory neural pathway that regulates hypertensive and natriuretic responses to acute changes in the composition of body fluid.
Forced internal desynchrony induces cardiometabolic alterations in adult rats.
Isis Gabrielli Barbieri de Oliveira,Marcos Divino Ferreira Junior,Paulo R. Lopes,Dhiogenes Balsanufo Taveira Campos,Marcos L. Ferreira-Neto,Eduardo Henrique Rosa Santos,Paulo Cezar de Freitas Mathias,Flávio Andrade Francisco,Bruna Del Vechio Koike,Carlos Henrique de Castro,André Henrique Freiria-Oliveira,Gustavo Rodrigues Pedrino,Rodrigo Mello Gomes,Daniel Alves Rosa +13 more
TL;DR: It is indicated that circadian timing desynchrony following the T22 protocol can induce cardiometabolic disruptions and early hepatic metabolism dysfunction can trigger other disorders, though additional studies are needed to clarify the causes.