Bruce F. Mackler
University of Texas Health Science Center at Houston
25 Papers
558 Citations
Bruce F. Mackler is an academic researcher from University of Texas Health Science Center at Houston. The author has contributed to research in topics: Antibody & Cytotoxic T cell. The author has an hindex of 16, co-authored 25 publications. Previous affiliations of Bruce F. Mackler include Harvard University & University of Maryland, Baltimore.
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Papers
Effects of Low- and High-Passage Influenza Virus Infection in Normal and Nude Mice
TL;DR: Two contrasting models of adaptation of type A Hong Kong influenza virus to mice suggest that thymus-dependent cells play a significant role in the inflammatory response to influenza virus infection and should prove useful for probing host-virus interactions which characterize influenza virus virulence.
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IgG subclasses in human periodontal disease. I. Distribution and incidence of IgG subclass bearing lymphocytes and plasma cells.
TL;DR: The evidence indicated that the majority of lymphocytes in mild gingivitis lesions lacked cytophilic IgG antibodies as well as Fc receptors, and the clinical stages of human perodontal disease are characterized by different populations of infiltrating lymphocytes.
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Quantitative analysis of cellular composition of human periapical granuloma
TL;DR: The cellular composition of 33 solid and cystic periapical granulomas was quantitated by a differential morphometric technique, and Macrophages were the predominant inflammatory cell, followed in descending numerical order by lymphocytes, plasma cells, and neutrophils.
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Antibody-producing cells in human periapical granulomas and cysts
TL;DR: Endodontic treatment of inflamed pulps did not alter the lesional distribution of immunoglobulin-positive cells compared with the distribution that prevailed in untreated patients, and there were no significant differences in immunoglOBulin distribution between the solid and cystic forms of the periapical lesions.
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Local and systemic immunoglobulins reactive to Bacteroides gingivalis in rapidly progressive and adult periodontitis.
TL;DR: Differences exist in the localized immunological responses of RP and AP patients to the periodontopathogen B. gingivalis that are not necessarily reflected in the systemic serum antibody responses of these patients.
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