Baile Ning
Guangzhou University of Chinese Medicine
18 Papers
30 Citations
Baile Ning is an academic researcher from Guangzhou University of Chinese Medicine. The author has contributed to research in topics: Autophagy & Medicine. The author has an hindex of 6, co-authored 8 publications.
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Papers
β-asarone improves learning and memory and reduces Acetyl Cholinesterase and Beta-amyloid 42 levels in APP/PS1 transgenic mice by regulating Beclin-1-dependent autophagy.
TL;DR: β-asarone treatment can improve the learning and memory abilities of APP/PS1 transgenic mouse by inhibiting Beclin-1-dependent autophagy via the PI3K/Akt/mTOR pathway.
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β-asarone inhibited cell growth and promoted autophagy via P53/Bcl-2/Bclin-1 and P53/AMPK/mTOR pathways in Human Glioma U251 cells
TL;DR: In this paper, the effect of β-asarone on inhibiting cell proliferation in human glioma U251 cells and detecting the effects of the inhibition on autophagy through the P53 signal pathway was studied.
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β-Asarone Regulates ER Stress and Autophagy Via Inhibition of the PERK/CHOP/Bcl-2/Beclin-1 Pathway in 6-OHDA-Induced Parkinsonian Rats.
TL;DR: It is deduced that β-asarone might regulate the ER stress-autophagy via inhibition of the PERK/CHOP/Bcl-2/Beclin-1 pathway in 6-OHDA-induced parkinsonian rats.
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β-Asarone Inhibits IRE1/XBP1 Endoplasmic Reticulum Stress Pathway in 6-OHDA-Induced Parkinsonian Rats
TL;DR: The team observed that β-asarone could improve the behavior of parkinsonian rats; increase the HVA, Dopacl, and 5-HIAA levels; and reduce α-synuclein levels, and deduced that β -asarone might be a potential candidate of medicine for clinical therapy of PD.
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Endoplasmic reticulum stress induced autophagy in 6-OHDA-induced Parkinsonian rats.
TL;DR: Investigation of the expressions of GRP78, CHOP, Beclin-1 and P62 in 6-OHDA-induced parkinsonian rat suggested that endoplasmic reticulum stress and autophagy occurred in6-OHda-induced rat, and ER stress might induce autophileagy.
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