Topic
Sympathectomy
About: Sympathectomy is a research topic. Over the lifetime, 3991 publications have been published within this topic receiving 77297 citations.
Papers published on a yearly basis
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Papers
TL;DR: The renal sympathetic nerves have been identified as a major contributor to the complex pathophysiology of hypertension in both experimental models and in humans and may be modulated by afferent signaling from renal sensory nerves.
Abstract: To the Editor: The renal sympathetic nerves have been identified as a major contributor to the complex pathophysiology of hypertension in both experimental models and in humans.1 Patients with essential hypertension generally have increased efferent sympathetic drive to the kidneys, as evidenced by elevated rates of renal norepinephrine spillover, defined as the amount of transmitter that escapes neuronal uptake and local metabolism and thus “spills over” into the circulation. Hypertension is also characterized by an increased rate of sympathetic-nerve firing, possibly modulated by afferent signaling from renal sensory nerves.2–4 A 59-year-old male patient with long-standing essential hypertension that was . . .
935 citations
TL;DR: The possiblity for the correctly diagnosed and treated patients to escape an otherwise impending death calls urgently for diffusion of the knowledge about the long Q-T syndrome.
888 citations
410 citations
TL;DR: S sympathetic neurocirculatory failure is defined as chronic, reproducible orthostatic hypotension associated with abnormal blood pressure responses in both phase II-L and phase IV of the Valsalva maneuver and neurochemical findings indicating decreased norepinephrine release, neuronal uptake, turnover, and synthesis in the heart could provide confirmation.
Abstract: Many patients with Parkinson disease—including all of those with sympathetic neurocirculatory failure—have evidence of cardiac sympathetic denervation. This finding suggests that loss of catecholam...
370 citations
TL;DR: A crucial experiment in the laboratory of neurology of the Boston City Hospital indicated that the efferent pathway for vasodilatation of cerebral arteries would be found in the facial nerve, and it was concluded that the pathway must leave the facial at the geniculate ganglion.
Abstract: A crucial experiment in the laboratory of neurology of the Boston City Hospital in which we cooperated with Cobb, Forbes and Finesinger indicated that the efferent pathway for vasodilatation of cerebral arteries would be found in the facial nerve. Returning to Montreal, we made a study 1 of the seventh cranial nerve and its connections, and found that there is a direct nerve bundle to the pericarotid plexus which leaves the facial nerve at the geniculate ganglion. Simultaneous physiologic 2 experimentation, carried out independently by Cobb and Finesinger, led them to conclude also that the pathway must leave the facial at the geniculate, as described in their contribution to be found in this issue, page 1243. Following previous work on the perivascular nerves of the brain (Penfield, 1931), we had found that complete sympathectomy did not markedly reduce the total number of perivascular nerve fibers. Therefore, having an excellent method
359 citations
Related Topics (5)
Performance Metrics
| Year | Papers |
|---|---|
| 2025 | 19 |
| 2024 | 37 |
| 2023 | 48 |
| 2022 | 69 |
| 2021 | 56 |
| 2020 | 43 |