TL;DR: In all patients with inferior wall infarction the isopotential map showed a minimum (area of negative potentials) on the inferior or right thoracic surface during the early portions of the QRS complex.
Abstract: Body surface isopotential maps obtained from 28 patients with old inferior wall myocardial infarction were compared with maps from 120 normal subjects. The 12 lead electrocardiogram of 8 of the 28 patients (29 percent) with inferior wall infarction was normal or showed only nondiagnostic ST-T wave abnormalities at the time the isopotential maps were obtained. In all patients with inferior wall infarction the isopotential map showed a minimum (area of negative potentials) on the inferior or right thoracic surface during the early portions of the QRS complex. This finding was observed in patients with normal or nonspecific abnormalities in the 12 lead electrocardiogram as well as those with QRS abnormalities. By contrast, the minimum during the early QRS complex in normal subjects was located on the right upper back and shoulder region. The body surface isopotential map may therefore be a useful clinical tool for detecting the approximately 25 percent of patients with previous inferior infarction whose electrocardiogram ultimately returns to normal or becomes nondiagnostic for the previous infarction. It contains information not available in the 12 lead electrocardiogram that may be useful in diagnosing various cardiac states in addition to inferior myocardial infarction.
TL;DR: Three approaches for detecting abnormalities in body surface potential maps recorded from patients with myocardial infarction were evaluated and it is found that the first approach is superior to the other two for detecting surface potential map abnormalities in patients withMyocardial Infarction.
TL;DR: In this paper, body surface potentials were recorded with the use of 63 unipolar leads in 45 patients with a non-Q-wave myocardial infarction (MI) (41 to 75 years old); 24 healthy adults, 42 patients with unstable angina, and 70 patients with Q-wave MI served as reference groups.
Abstract: Background—Potential losses caused by stable non–Q-wave myocardial infarction (MI) are too small to diagnose with the use of standard ECG. The aim of the present study was to obtain accurate diagnostic criteria for this prognostically important disease with the help of body surface mapping. Methods and Results—Body surface potentials were recorded with the use of 63 unipolar leads in 45 patients with a non–Q-wave MI (41 to 75 years old); 24 healthy adults, 42 patients with unstable angina, and 70 patients with Q-wave MI served as reference groups. Qualitative pathological features of the isopotential maps, such as onset time and site and magnitude of the first right-anterior/anterior minimum, as well as pathological negativities at that time, were defined in non–Q-wave MI cases. These features, which account for the activation sequence and the body surface projections of specific cardiac regions (Selvester classification), showed a 91% sensitivity and an 88% specificity for the detection of non–Q-wave MI....
TL;DR: The isopotential area of VOTAs originating from the RVOT, as compared to the other sites, spread more elliptically and slowly, which can provide useful information and efficient strategy for VOTA ablation.
Abstract: Ventricular outflow tract arrhythmias (VOTAs) can be successfully treated by catheter ablation. However, it is sometimes difficult to differentiate the origin of VOTAs between the right ventricular outflow tract (RVOT) and the other sites, leading to a long fluoroscopy time and unnecessary radiofrequency applications. This study aimed to clarify distinguishable characteristics of the propagation pattern obtained from non-contact mapping (NCM) for VOTA ablation. Consecutive 45 patients with VOTAs who underwent catheter ablation using the NCM system were included in this study. We analyzed an isopotential map on three-dimensional geometry of the RVOT obtained from the virtual unipolar electrograms (VUEs) and assessed mapping data of the isopotential area with an initial negative VUE of −1 mV. Successful ablation was achieved from the endocardial RVOT in 34 patients (RVOT group) and the non-RVOT in 11 (non-RVOT group). Major and minor axis diameters of the isopotential area did not significantly differ between the two groups. However, a ratio of major/minor axis diameter was greater in the RVOT group (1.9 ± 0.1 versus 1.3 ± 0.1; P < 0.001). In addition, the propagation velocity defined as an increase of the isopotential area per millisecond was significantly slower in the RVOT group (2.2 ± 0.4 versus 4.2 ± 0.7 mm2/ms; P = 0.02). The isopotential area of VOTAs originating from the RVOT, as compared to the other sites, spread more elliptically and slowly. The propagation pattern obtained from NCM can provide useful information and efficient strategy for VOTA ablation.