About: Dehydroascorbic acid transport is a research topic. Over the lifetime, 22 publications have been published within this topic receiving 1994 citations.
TL;DR: Ascorbate is proposed as a neuromodulator of glutamatergic, dopaminergic, cholinergic, and GABAergic transmission and related behaviors, posited to have potential therapeutic roles against ischemic stroke, Alzheimer's disease, Parkinson’s disease, and Huntington's disease.
TL;DR: GLUT1 and GLUT3 isoforms are the specific glucose transporter isoforms which mediate DHA transport and subsequent accumulation of AA according to Xenopus laevis oocyte expression system studies.
TL;DR: It is indicated that the insulin-sensitive transporter GLUT4 transports DHA in both rat adipocytes and Xenopus oocytes, and Alterations of this mechanism in diabetes could have clinical implications for ascorbate utilization.
TL;DR: New information is provided that ascorbate and dehydroascorbic acid are transported into human neutrophils and fibroblasts by two distinct mechanisms and that the compound available for intracellular utilization is asCorbate.
TL;DR: The data indicate that genistein inhibits the transport of dehydroascorbic acid and hexoses by directly interacting with the hexose transporter GLUT1 and interfering with its transport activity, rather than as a consequence of its known ability to inhibit protein-tyrosine kinases.