Journal Article10.1097/00007632-199310000-00015
Myofascial trigger points show spontaneous needle EMG activity.
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TL;DR: The authors hypothesize that TrPs are caused by sympathetically activated intrafusal contractions.
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Abstract: Monopolar needle electromyogram (EMG) was recorded simultaneously from trapezius myofascial trigger points (TrPs) and adjacent nontender fibers (non-TrPs) of the same muscle in normal subjects and in two patient groups, tension headache and fibromyalgia. Sustained spontaneous EMG activity was found in the 1-2 mm nidus of all TrPs, and was absent in non-TrPs. Mean EMG amplitude in the patient groups was significantly greater than in normals. The authors hypothesize that TrPs are caused by sympathetically activated intrafusal contractions.
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Citations
Myofascial trigger points alter the modular control during the execution of a reaching task: a pilot study
TL;DR: The present results suggest that the increased weighting coefficients in presence of TPs are associated with an alteration of the modular motor control without affecting the dimensionality of motor modules for each individual and reciprocal inhibition.
Myelinated Afferents Are Involved in Pathology of the Spontaneous Electrical Activity and Mechanical Hyperalgesia of Myofascial Trigger Spots in Rats.
TL;DR: It is concluded that proliferated myelinated afferents existed at MTrSs, which were closely related to pathology of SEA and mechanical hyperalgesia of MtrSs.
Objective Measurement of Tissue Tension in Myofascial Trigger Point Areas Before and During the Administration of Anesthesia With Complete Blocking of Neuromuscular Transmission
Johannes Buchmann,Beate Neustadt,Katharina Buchmann-Barthel,Soeren Rudolph,Thomas Klauer,Olaf Reis,Ulrich Smolenski,Hella Buchmann,Klaus F. Wagner,Frank Haessler +9 more
TL;DR: An increased muscle tension in MTPs, and not a primary local inflammation with enhanced viscoelasticity, was the main result of the investigation, and this increased muscular tension in the taut band with an MTP is interpreted as increased spinal segmental excitability.
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Intrapericardial algogenic chemicals evoke cardiac-somatic motor reflexes in rats.
TL;DR: The aim is to develop an animal model to study muscle changes resulting from cardiac dysfunction and to investigate possible links between muscle hyperalgesia and EMG activities observed with ischemic heart diseases.
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Musculoskeletal Ultrasonography in CRPS: Assessment of Muscles Before and After Motor Function Recovery with Dry Needling as the Sole Treatment.
TL;DR: Myofascial pathology of co-contraction appears to cause CDC of CRPS and probable ischemic loss of myoarchitecture in 44 patients treated between December 2005 and December 2014.
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