Interactions between sleep, stress, and metabolism: From physiological to pathological conditions

TL;DR: In this article , a review provides detailed insight into the complex crosstalk between stress, sleep, and circadian rhythm, their relationship with the body's homeostasis, and the consequences of circadian dysregulation.

TL;DR: In this article, the authors investigated the association between time-restricted feeding (TRF) and mental health outcomes including perceived stress, depressive symptoms, and sleep quality assessed in a cohort of southern Italian adults.

TL;DR: In this paper, work stress and impaired sleep are established risk factors for cardiovascular disease (CVD) among healthy individuals, but their impact on hypertensive workers is largely unknown, and their effect on CVD risk is unknown.

TL;DR: In this article , the authors found that CBD had greater anxiolytic effects without prominent effects on sleep in healthy and non-cannabis using clinical populations, and an inverted U-shaped dose relationship and CBD ratio to THC in combined treatments likely moderated these effects.

TL;DR: Differences in leptin and ghrelin are likely to increase appetite, possibly explaining the increased BMI observed with short sleep duration, and changes in appetite regulatory hormones with sleep curtailment may contribute to obesity.

TL;DR: In this study, 12 young, healthy, normal-weight men exhibited reductions in the satiety hormone leptin, increases in the hunger hormone ghrelin, and increases in hunger after 2 nights of only 4 hours of sleep compared with after two nights of 10 hours ofSleep, suggesting inadequate sleep seems to influence the hormones that regulate satiety and hunger.

TL;DR: In this article, a meta-analysis was carried out to assess whether the evidence supports the presence of a relationship between short sleep duration and obesity at different ages, and to obtain a quantitative estimate of the risk in order to assess the consistency and potential public health relevance.

TL;DR: Increased adipocyte 11β HSD-1 activity may be a common molecular etiology for visceral obesity and the metabolic syndrome.