Immediate treatment with propranolol decreases posttraumatic stress disorder two months after trauma

TL;DR: The neurobiology of both anxiety and fear can be found in the brain this paper, which can serve to be adaptive in shaping decisions and behaviors related to survival of an organism, but when excessive, or pathological, or triggered inappropriately, fear and anxiety form the basis of a variety of anxiety disorders such as generalized anxiety disorder (GAD) or obsessive-compulsive disorder (OCD).

TL;DR: This review aims to demonstrate how an understanding of the brain mechanisms involved in memory provides a basis for reconceptualizing some mental disorders; refining existing therapeutic tools; and designing new ones for targeting processes that maintain these disorders.

TL;DR: This chapter addresses questions regarding the complex etiology of PTSD, and the relative strength of discernable biological indicators of the disorder, and defines the constructs of multicausality, equifinality, and multifinality and evaluates their main implication for studies of PTSD.

TL;DR: The Mini International Neuropsychiatric Interview (MINI) as mentioned in this paper is a short diagnostic structured interview (DSI) developed in France and the United States to explore 17 disorders according to Diagnostic and Statistical Manual (DSM)-III-R diagnostic criteria.

TL;DR: The results supported the validity and reliability of the MINI and the application of short structured interviews in clinical and research settings is discussed.

TL;DR: The impairment of propranolol on memory of the emotional story was not due either to reduced emotional responsiveness or to nonspecific sedative or attentional effects, which support the hypothesis that enhanced memory associated with emotional experiences involves activation of the β-adrenergic system.

TL;DR: These pilot results suggest that acute, posttrauma propranolol may have a preventive effect on subsequent PTSD.