Journal Article10.1016/J.BRAINRES.2006.09.009
Curcumin reverses the effects of chronic stress on behavior, the HPA axis, BDNF expression and phosphorylation of CREB.
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TL;DR: The results provide compelling evidence that the behavioral effects of curcumin in chronically stressed animals, and by extension humans, may be related to their modulating effects on the HPA axis and neurotrophin factor expressions.
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About: This article is published in Brain Research. The article was published on 29 Nov 2006. The article focuses on the topics: Brain-derived neurotrophic factor & CREB.
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Citations
Enrichment Effects on Adult Cognitive Development: Can the Functional Capacity of Older Adults Be Preserved and Enhanced?
TL;DR: The available evidence suggests that activities can postpone decline, attenuate decline, or provide prosthetic benefit in the face of normative cognitive decline, while at the same time indicating that late-life cognitive changes can result in curtailment of activities.
Brain-Derived Neurotrophic Factor: A Key Molecule for Memory in the Healthy and the Pathological Brain.
TL;DR: It is proposed that, although BDNF may not be a valid biomarker for neurodegenerative/neuropsychiatric diseases, it could be thought of as a marker that specifically relates to the occurrence and/or progression of the mnemonic symptoms that are common to many pathological conditions.
Bipolar and Major Depressive Disorder: Neuroimaging the Developmental-Degenerative Divide
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TL;DR: It is suggested that environmental stress and genetic risk variants interact with each other in a complex manner to alter neural circuitry and precipitate illness.
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Neurobiology of chronic mild stress: parallels to major depression.
TL;DR: The chronic mild stress paradigm evokes an array of neurobiological changes that mirror those seen in depressive disorders and may be a suitable tool to investigate novel systems that could be disturbed in depression, and thus aid in the development of novel targets for the treatment of depression.
425
Neuroplasticity mediated by altered gene expression.
TL;DR: This review summarizes some of the major transcriptional mechanisms that are thought to underlie neuronal and behavioral plasticity in the brain.
349
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