Copper nanoparticles and copper ions promote horizontal transfer of plasmid-mediated multi-antibiotic resistance genes across bacterial genera.

TL;DR: In this paper, the authors provide a research roadmap to address existing challenges in AMR control via wastewater treatment, including inconsistencies in ARG reporting units, lack of ARG/ARB monitoring surrogates, and lack of a standardised protocol for determining ARG removal via wastewater treatments.

TL;DR: In this paper , the authors compared the killing mechanisms and structure-activity relationships of antibiotics and antimicrobial nanomaterials, and then focused on nano-microbe interactions to elucidate the impacts of molecular initiating events on AMR evolution.

TL;DR: Evaluation of prevalence/persistence and horizontal transfer of iARGs and extracellular ARGs (eARGs) in different environments, and advanced mitigation strategies in wastewater treatment plants (WWTPs) for preventing the spread of antibiotic resistance in the environment are explored.

TL;DR: A comprehensive review of the effects and intrinsic mechanisms of EPs, including microplastics, engineered nanomaterials, disinfection byproducts, pharmaceuticals, and personal care products, on the occurrence and dissemination of ARGs is presented in this paper.

TL;DR: Investigating the multiple responses of Pseudomonas aeruginosa to the exposure of copper oxide nanoparticles shows that CuO NPs induce lysogenic bacteriophage, which might render defective within a bacterial host.

TL;DR: Western blotting demonstrated that the apparently mutual enhancement of repression is not due simply to elevation of repressor level by the presence of the second protein, suggesting that the basis for cooperativity is interaction between KorB and TrbA bound at their respective operators.

TL;DR: It is found AgNPs stimulated the proliferation of D. vulgaris, rather than exerting inhibitory or biocidal effects, which implies that the presence of AgNP in sewage may affect SRB abundance in sewer networks.

TL;DR: It is demonstrated that the RpoS general stress regulator prevents oxidative stress-mediated DNA damage formation in E. coli, suggesting that E. Escherichia coli is more of an exception than a paradigm for the physiological response to antibiotics sub-MIC.