Circulation to Skeletal Muscle

TL;DR: The sections in this article are: Basal Vascular Resistance and Autoregulation, Role of Myogenic Mechanism in Reactive Hyperemia, and Conclusions.

Abstract: The sections in this article are: 1 Basal Vascular Resistance and Autoregulation 2 Role of Myogenic Mechanism in Reactive Hyperemia 3 Role of Myogenic Mechanism in Exercise Hyperemia 4 Types of Skeletal Muscle Fibers 5 Muscle Blood Flow During Exercise 5.1 Oxygen Lack 5.2 Lactic Acid, pH, and CO2 5.3 Potassium 5.4 Inorganic Phosphate 5.5 Osmolarity 5.6 Prostaglandins 5.7 Adenosine and Adenine Nucleotides 5.8 Intrinsic Neurons and Exercise Dilatation 5.9 General Conclusions 6 Mechanical Hindrance to Blood Flow in Skeletal Muscle 7 Reactive Hyperemia 7.1 Are Exercise and Reactive Hyperemia Due to the Same Mechanism(s)? 8 Propagated Vasodilatation 9 Sympathetic Vasomotor Outflow 10 Noradrenergic Innervation of Resistance Vessels in Muscles 10.1 Neuroeffector Junction 10.2 α-Adrenergic Activation 11 Factors that Modulate Transmitter Output From Sympathetic Nerve Endings 11.1 Metabolic Action 11.2 Neurohumoral Action 12 Reflex Regulation of Skeletal Muscle Resistance Vessels by Sympathetic Noradrenergic Nerves 12.1 Carotid and Aortic Baroreflex 12.2 Carotid and Aortic Chemoreflex 12.3 Reflexes From the Heart and Lungs 12.4 Diving Reflex 12.5 Reflex From Receptors in Skeletal Muscles 13 Rhythmic Exercise 14 Isometric Exercise 15 Influence of Sympathetic Noradrenergic Fibers on the Blood Flow to Active Muscles 16 β2-Adrenergic Activation 17 Cholinergic Vasodilator Nerves 17.1 Role of Cholinergic Nerves in the Carotid Baroreflex 17.2 Cholinergic Vasodilatation in Humans 18 Histaminergic Vasodilatation 18.1 Role of Histamine in the Vasodilatation Resulting From Reflex Inhibition of Sympathetic Outflow 19 Conclusions