Circadian Time-Place Learning in Mice Depends on Cry Genes

TL;DR: Evidence for non-transcriptional and network based circadian rhythmicity provides precedence, but any such mechanisms are likely to interact closely with known circadian clock genes, and some important double and triple clock gene knockouts remain to be phenotyped for food entrainment.

TL;DR: Together, these studies suggest that common mechanisms underlie circadian rhythmicity and long-term memory formation.

TL;DR: Criteria for distinguishing among formally distinct mechanisms by which animals might anticipate a daily meal are reviewed, and procedural variables that can affect the expression of food‐anticipatory rhythms in neurobiologically intact or compromised animals are identified.

TL;DR: The main strands of the literature on the feedbacks between environmental predictability and animal movement are synthesised and how they may react to anthropogenic disruption is discussed, leading to unexpected threats for wildlife and the environment.

TL;DR: It is shown that mice lacking the Cry1 or Cry2 protein display accelerated and delayed free-running periodicity of locomotor activity, respectively, which suggests that, in addition to a possible photoreceptor and antagonistic clock-adjusting function, both proteins are essential for the maintenance of circadian rhythmicity.

TL;DR: These results indicate that mice lacking cryptochromes are not simply a genetic model of circadian arrhythmicity in rodents and functionally implicate cryptochromaes in the homeostatic regulation of sleep.

TL;DR: It is shown that rats have a detailed representation of remembered events and meet behavioral criteria for episodic-like memory, and it is demonstrated that rats selectively encode the content of episodi-like memories.

TL;DR: In this article, kristallisieren sich in der Beurteilung der untersuchten biologischen Rhythmen zwei Anschauungen heraus.