Book Chapter10.1016/B978-0-12-385095-9.00004-X
Chapter 4 – EPF
Tatsuo Kakimoto
- 01 Jan 2013
pp 20-23
About: The article was published on 01 Jan 2013.
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References
Stomatal Development and Patterning Are Regulated by Environmentally Responsive Mitogen-Activated Protein Kinases in Arabidopsis
TL;DR: It is reported that Arabidopsis MITOGEN-ACTIVATED PROTEIN KINASE3 (MPK3) and MPK6, two environmentally responsive mitogen-activated protein kinases (MAPK) and their upstream MAPK kinases, MKK4 and MKK5, are key regulators of stomatal development and patterning.
Stomatal Patterning and Differentiation by Synergistic Interactions of Receptor Kinases
TL;DR: This work reports that three ERECTA (ER)–family leucine-rich repeat–receptor-like kinases (LRR-RLKs) together control stomatal patterning, with specific family members regulating the specification ofStomatal stem cell fate and the differentiation of guard cells.
612
Transcription factor control of asymmetric cell divisions that establish the stomatal lineage
TL;DR: It is demonstrated that SPCH and two paralogues are successively required for the initiation, proliferation and terminal differentiation of cells in the stomatal lineage in Arabidopsis thaliana, highlighting a conserved use of closely related bHLHs for cell fate specification and differentiation.
587
Stomatal development and pattern controlled by a MAPKK kinase.
TL;DR: In this paper, the Arabidopsis mitogen-activated protein kinase kinase (YODA) gene was used to identify potential stomatal regulatory genes, and a putative transcription factor from this set was shown to regulate the developmental behavior of stomata precursors.
563
SCREAM/ICE1 and SCREAM2 Specify Three Cell-State Transitional Steps Leading to Arabidopsis Stomatal Differentiation
Masahiro M. Kanaoka,Lynn Jo Pillitteri,Hiroaki Fujii,Yuki Yoshida,Naomi L. Bogenschutz,Junji Takabayashi,Jian-Kang Zhu,Keiko U. Torii +7 more
TL;DR: This work identifies two paralogous proteins, SCREAM (SCRM) and SCRM2, which directly interact with and specify the sequential actions of SPCH, MUTE, and FAMA and suggests a model strikingly similar to cell-type differentiation in animals.
556