Central cholinergic pathways in the rat: an overview based on an alternative nomenclature (Ch1-Ch6).

TL;DR: This study, using fMRI in conjunction with masked stimulus presentations, represents an initial step toward determining the role of the amygdala in nonconscious processing.

TL;DR: Although AT(1) receptors mediate most of the known actions of Ang II, the AT(2) receptor contributes to the regulation of blood pressure and renal function and the development of specific nonpeptide receptor antagonists has led to major advances in the physiology, pharmacology, and therapy of the renin-angiotensin system.

TL;DR: It is suggested that fimbrial transections resulted in retrograde degeneration of cholinergic septo-hippocampal neurons and that NGF treatment strongly attenuated this lesion-induced degeneration.

TL;DR: Evidence is presented that the nucleus basalis-neocortical cholinergic system contributes greatly to visual attentional function, but not to mnemonic processes per se, and it is suggested that nucleus basali-amygdala cholinerential projections have a role in the retention of affective conditioning while brainstem cholinery projections to the thalamus and midbrain dopamine neurons affect basic arousal processes.

TL;DR: It is suggested that small oral doses of physostigmin combined with lecithin ingestion have therapeutic benefit for some patients with Alzheimer disease.

TL;DR: The results indicate that the acute augmentation of cholinergic activity in some patients with Alzheimer's disease can partially reverse the memory deficit of that disorder and may provide an approach to the eventual therapy of this condition.

TL;DR: The features of cortically projecting intralaminar neurons that relay MRF activity fit in well with their hypothesized role in the tonic activation processes that characterize both W and D states as compared to S sleep, and several lines of evidence suggest that sustained hyperpolarization prevails in intralamar neurons during S sleep.

TL;DR: These results provide further support for the idea that the amnesia produced by scopolamine is due to a direct dysfunction in central cholinergic mechanisms, and is not due to some general effect involving arousal, attention, etc.