Anomalous rectification in the squid giant axon injected with tetraethylammonium chloride.

TL;DR: The sections in this article are: Electrical Properties of Ganglion Cells, Classification of Smooth Muscles, Cellular Basis of some of the Actions of the Autonomic Nervous System, and Conclusion.

Abstract: Abstract The sections in this article are: Resting Membrane Potential Contribution of Diffusion Potential Contribution of Electrogenic Pumps Excitable Events in Gastric Muscles Gastric Slow Waves Action Potentials Electrical Arrhythmias Electrical Events of Fundus Electrical Activity of Muscularis Mucosa Electrical Activity of Amphibian Stomach Excitation‐Contraction Coupling Mechanical Threshold Anatomical Variation of Voltage‐Tension Relationship Voltage Dependence of Ca 2+ Channels Electropharmacology of Endogenous Substances Adrenergic Agonists Cholinergic Agonists Peptides Prostaglandins Propagation of Electrical Events Cell‐to‐Cell Coupling Passive Characteristics of Gastric Syncytium Measurements of Conduction Velocity Use of Conduction‐ Velocity Measurements to Determine Site of Slow‐Wave Origin Myogenic Regulation of Propagation Models of Excitability and Propagation Relaxation‐Oscillator Models Cable Models Directions for Future Research

TL;DR: This chapter describes the plasma membrane of nerve cells, which is found in the central nervous system of mammals and in the peripheral nervous system, which extends throughout the body.

TL;DR: Acetylcholine, and to a smaller extent histamine, are less dependent upon the presence of extracellular calcium, and may be capable of releasing calcium sequestered within the cell; acetyl choline appears to be more effective in releasing sequestered calcium.

TL;DR: The identity of the ions which carry the various phases of the membrane current is chiefly concerned with sodium ions, since there is much evidence that the rising phase of the action potential is caused by the entry of these ions.

TL;DR: The importance of ionic movements in excitable tissues has been emphasized by a number of recent experiments which are consistent with the theory that nervous conduction depends on a specific increase in permeability which allows sodium ions to move from the more concentrated solution outside a nerve fibre to the more dilute solution inside it.

TL;DR: This paper contains a further account of the electrical properties of the giant axon of Loligo and deals with the 'inactivation' process which gradually reduces sodium permeability after it has undergone the initial rise associated with depolarization.