Example of Annals of Clinical Biochemistry format
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Example of Annals of Clinical Biochemistry format Example of Annals of Clinical Biochemistry format Example of Annals of Clinical Biochemistry format Example of Annals of Clinical Biochemistry format
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Example of Annals of Clinical Biochemistry format Example of Annals of Clinical Biochemistry format Example of Annals of Clinical Biochemistry format Example of Annals of Clinical Biochemistry format
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open access Open Access

Annals of Clinical Biochemistry — Template for authors

Publisher: SAGE
Categories Rank Trend in last 3 yrs
Clinical Biochemistry #66 of 113 down down by 2 ranks
journal-quality-icon Journal quality:
Medium
calendar-icon Last 4 years overview: 322 Published Papers | 1242 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 22/07/2020
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Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

2.044

8% from 2018

Impact factor for Annals of Clinical Biochemistry from 2016 - 2019
Year Value
2019 2.044
2018 1.893
2017 1.983
2016 2.024
graph view Graph view
table view Table view

3.9

22% from 2019

CiteRatio for Annals of Clinical Biochemistry from 2016 - 2020
Year Value
2020 3.9
2019 3.2
2018 3.3
2017 3.4
2016 3.4
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 8% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 22% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

0.6

6% from 2019

SJR for Annals of Clinical Biochemistry from 2016 - 2020
Year Value
2020 0.6
2019 0.566
2018 0.624
2017 0.634
2016 0.588
graph view Graph view
table view Table view

0.81

0% from 2019

SNIP for Annals of Clinical Biochemistry from 2016 - 2020
Year Value
2020 0.81
2019 0.807
2018 0.78
2017 0.845
2016 0.799
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has increased by 6% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 0% in last years.
  • This journal’s SNIP is in the top 10 percentile category.
Annals of Clinical Biochemistry

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SAGE

Annals of Clinical Biochemistry

One of the world's foremost in its field, Annals publishes fully refereed papers of international authorship that contribute to existing knowledge in all fields of clinical biochemistry, especially that pertaining to the understanding, diagnosis and treatment of human disease....... Read More

Medicine

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Last updated on
22 Jul 2020
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ISSN
0004-5632
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Impact Factor
Medium - 0.935
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Open Access
No
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Sherpa RoMEO Archiving Policy
Green faq
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Endnote Style
Download Available
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Bibliography Name
SageV
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Citation Type
Numbered (Superscripted)
25
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Bibliography Example
Blonder GE, Tinkham M and Klapwijk TM. Transition from metallic to tunneling regimes in superconducting microconstrictions: Excess current, charge imbalance, and supercurrent conversion. Phys. Rev. B 1982; 25(7): 4515–4532. URL 10.1103/PhysRevB.25.4515.

Top papers written in this journal

Journal Article DOI: 10.1177/000456326900600108
Determination of Glucose in Blood Using Glucose Oxidase with an Alternative Oxygen Acceptor

Abstract:

the oxygen acceptors originally used were 0 tolidine, benzidine and o-dianisidine. It has since been established that these three substances are carcinogens and many alternative oxygen acceptors have been suggested. Any dye showing oxidation-reduction properties or any dye formed by oxidation, such as those used in colour pho... the oxygen acceptors originally used were 0 tolidine, benzidine and o-dianisidine. It has since been established that these three substances are carcinogens and many alternative oxygen acceptors have been suggested. Any dye showing oxidation-reduction properties or any dye formed by oxidation, such as those used in colour photography, are potentially useful but it is obviously advantageous to use reagents which have high stability. For manual work on blood a two-solution technique is desirable, one solution being used to precipitate the protein and the other to develop the colour. The development of such a method will now be described. In the determination of phosphatase, use is made of the fact that phenol in the presence of an oxidising reagent gives a purple colour with 4-amino phenazone. The possibility that the H.Oz released in the reaction of glucose oxidase with glucose could act as the oxidising agent was investigated and it was found that the system of phenol and 4-amino phenazone is well suited to the determination of glucose. By suitable adjustment of conditions the colour develops completely in 10 minutes, being stable thereafter for at least 30 minutes. Using a single-solution phosphotungstic acid precipitant containing phenol to precipitate blood protein the only other solution required is one containing glucose oxidase, peroxidase and 4-amino phenazone. These solutions contain azide as preservative; azide has no effect on the rate of colour development. In the micro and macro automated methods, the two solutions required are a diluent containing 4-amino phenazone and a colour reagent containing glucose oxidase, peroxidase and phenol. read more read less
5,109 Citations
Journal Article DOI: 10.1177/0004563213480712
Classification of acute pancreatitis – 2012: revision of the Atlanta classification and definitions by international concensus
Helen Kinns1

Abstract:

Background and objective The Atlanta classification of acute pancreatitis enabled standardised reporting of research and aided communication between clinicians. Deficiencies identified and improved understanding of the disease make a revision necessary. Methods A web-based consultation was undertaken in 2007 to ensure wide pa... Background and objective The Atlanta classification of acute pancreatitis enabled standardised reporting of research and aided communication between clinicians. Deficiencies identified and improved understanding of the disease make a revision necessary. Methods A web-based consultation was undertaken in 2007 to ensure wide participation of pancreatologists. After an initial meeting, the Working Group sent a draft document to 11 national and international pancreatic associations. This working draft was forwarded to all members. Revisions were made in response to comments, and the web-based consultation was repeated three times. The final consensus was reviewed, and only statements based on published evidence were retained. Results The revised classification of acute pancreatitis identified two phases of the disease: early and late. Severity is classified as mild, moderate or severe. Mild acute pancreatitis, the most common form, has no organ failure, local or systemic complications and usually resolves in the first week. Moderately severe acute pancreatitis is defined by the presence of transient organ failure, local complications or exacerbation of co-morbid disease. Severe acute pancreatitis is defined by persistent organ failure, that is, organ failure >48 h. Local complications are peripancreatic fluid collections, pancreatic and peripancreatic necrosis (sterile or infected), pseudocyst and walled-off necrosis (sterile or infected). We present a standardised template for reporting CT images. Conclusions This international, web-based consensus provides clear definitions to classify acute pancreatitis using easily identified clinical and radiologic criteria. The wide consultation among pancreatologists to reach this consensus should encourage widespread adoption. read more read less
3,546 Citations
Journal Article DOI: 10.1177/000456328302000601
Salivary Cortisol: A Better Measure of Adrenal Cortical Function than Serum Cortisol
Ross F. Vining1, Robynne A. McGinley1, Joseph J Maksvytis1, Kian Y Ho1

Abstract:

Salivary cortisol concentration was found to be directly proportional to the serum unbound cortisol concentration both in normal men and women and in women with elevated cortisol-binding globulin (CBG). The correlation was excellent in dynamic tests of adrenal function (dexamethasone suppression, ACTH stimulation), in normals... Salivary cortisol concentration was found to be directly proportional to the serum unbound cortisol concentration both in normal men and women and in women with elevated cortisol-binding globulin (CBG). The correlation was excellent in dynamic tests of adrenal function (dexamethasone suppression, ACTH stimulation), in normals and patients with adrenal insufficiency, in tests of circadian variation and randomly collected samples. Women in the third trimester of normal pregnancy exhibited elevated salivary cortisol throughout the day. The relationship between salivary and serum total cortisol concentration was markedly non-linear with a more rapid increase in salivary concentration once the serum CBG was saturated. The rate of equilibrium of cortisol between blood and saliva was very fast, being much less than 5 minutes. These data, combined with a simple, stress-free, non-invasive collection procedure, lead us to suggest that salivary cortisol is a more appropriate measure for the clinical assessment of adrenocortical function than is serum cortisol. read more read less
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723 Citations
open accessOpen access Journal Article DOI: 10.1177/0004563218759371
The bone remodelling cycle.
Julia Kenkre1, Jhd Bassett1

Abstract:

The bone remodelling cycle replaces old and damaged bone and is a highly regulated, lifelong process essential for preserving bone integrity and maintaining mineral homeostasis. During the bone remodelling cycle, osteoclastic resorption is tightly coupled to osteoblastic bone formation. The remodelling cycle occurs within the... The bone remodelling cycle replaces old and damaged bone and is a highly regulated, lifelong process essential for preserving bone integrity and maintaining mineral homeostasis. During the bone remodelling cycle, osteoclastic resorption is tightly coupled to osteoblastic bone formation. The remodelling cycle occurs within the basic multicellular unit and comprises five co-ordinated steps; activation, resorption, reversal, formation and termination. These steps occur simultaneously but asynchronously at multiple different locations within the skeleton. Study of rare human bone disease and animal models have helped to elucidate the cellular and molecular mechanisms that regulate the bone remodelling cycle. The key signalling pathways controlling osteoclastic bone resorption and osteoblastic bone formation are receptor activator of nuclear factor-κB (RANK)/RANK ligand/osteoprotegerin and canonical Wnt signalling. Cytokines, growth factors and prostaglandins act as paracrine regulators of the cycle, whereas endocrine regulators include parathyroid hormone, vitamin D, calcitonin, growth hormone, glucocorticoids, sex hormones, and thyroid hormone. Disruption of the bone remodelling cycle and any resulting imbalance between bone resorption and formation leads to metabolic bone disease, most commonly osteoporosis. The advances in understanding the cellular and molecular mechanisms underlying bone remodelling have also provided targets for pharmacological interventions which include antiresorptive and anabolic therapies. This review will describe the remodelling process and its regulation, discuss osteoporosis and summarize the commonest pharmacological interventions used in its management. read more read less
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535 Citations
open accessOpen access Journal Article DOI: 10.1258/ACB.2009.009222
The detection and prevention of errors in laboratory medicine.

Abstract:

The last few decades have seen a significant decrease in the rates of analytical errors in clinical laboratories. Evidence demonstrates that pre- and post-analytical steps of the total testing process (TTP) are more error-prone than the analytical phase. Most errors are identified in pre-pre-analytic and post-post-analytic st... The last few decades have seen a significant decrease in the rates of analytical errors in clinical laboratories. Evidence demonstrates that pre- and post-analytical steps of the total testing process (TTP) are more error-prone than the analytical phase. Most errors are identified in pre-pre-analytic and post-post-analytic steps outside of the laboratory. In a patient-centred approach to the delivery of health-care services, there is the need to investigate, in the TTP, any possible defect that may have a negative impact on the patient. In the interests of patients, any direct or indirect negative consequence related to a laboratory test must be considered, irrespective of which step is involved and whether the error depends on a laboratory professional (e.g. calibration/testing error) or non-laboratory operator (e.g. inappropriate test request, error in patient identification and/or blood collection). Patient misidentification and problems communicating results, which affect the delivery of diagnostic services, are recognized as the main goals for quality improvement. International initiatives aim at improving these aspects. Grading laboratory errors on the basis of their seriousness should help identify priorities for quality improvement and encourage a focus on corrective/preventive actions. It is important to consider not only the actual patient harm sustained but also the potential worst-case outcome if such an error were to reoccur. The most important lessons we have learned are that system theory also applies to laboratory testing and that errors and injuries can be prevented by redesigning systems that render it difficult for all health-care professionals to make mistakes. read more read less
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436 Citations
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Frequently asked questions

1. Can I write Annals of Clinical Biochemistry in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Annals of Clinical Biochemistry guidelines and auto format it.

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Yes, the template is compliant with the Annals of Clinical Biochemistry guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Annals of Clinical Biochemistry?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Annals of Clinical Biochemistry citation style.

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Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Annals of Clinical Biochemistry.

5. Can I use a manuscript in Annals of Clinical Biochemistry that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Annals of Clinical Biochemistry that you can download at the end.

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7. Where can I find the template for the Annals of Clinical Biochemistry?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Annals of Clinical Biochemistry's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

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Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

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After writing your paper autoformatting in Annals of Clinical Biochemistry, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Annals of Clinical Biochemistry's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Annals of Clinical Biochemistry?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Annals of Clinical Biochemistry. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Annals of Clinical Biochemistry?

The 5 most common citation types in order of usage for Annals of Clinical Biochemistry are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Annals of Clinical Biochemistry?

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16. Can I download Annals of Clinical Biochemistry in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Annals of Clinical Biochemistry Endnote style according to Elsevier guidelines.

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