Advanced oxidation processes for water disinfection: Features, mechanisms and prospects

Electrified membranes (EMs) have the potential to address inherent limitations of conventional membrane technologies. Recent studies have demonstrated that EMs exhibit enhanced functions beyond sep...

Electrified Membranes for Water Treatment Applications

Biosynthesis of copper nanoparticles using Shewanella loihica PV-4 with antibacterial activity: Novel approach and mechanisms investigation.

Electrochemical oxidation of p-nitrophenol was examined using differentanodic materials, including T/boron-doped diamond (BDD), Ti/SnO2-Sb/PbO2, and Ti/SnO2-Sb anodes. The results demonstrated that Ti/BDD anodes had a much stronger mineralization performance than the other two anodes. Furthermore, it was found that hydroxyl radicals could mainly exist as free hydroxyl radicals at BDD anodes, which could react with organic compounds effectively. This implied that the dominant mechanism for a much higher mineralization capacity of BDD anodes would be attributed to the existence of free hydroxyl radicals in the BDD anode cell rather than adsorbed hydroxyl radicals on the BDD anode. To further corroborate this hypothesis, electrochemical oxidation of p-substituted phenols (p-nitrophenol, p-hydroxybenzaldehyde, phenol, p-cresol, and p-methoxyphenol) was examined at the Ti/BDD, Ti/SnO2-Sb/ PbO2, and Ti/SnO2-Sb anodes, respectively. The study revealed that for Ti/BDD electrodes, the degradation rate of p-substituted phenols (k) increased with the increase of Hammett's constant (sigma), which confirmed the dominance of free hydroxyl radicals at BDD anodes and its effective reaction with organics therein. For Ti/SnO2-Sb/PbO2 electrodes, the degradation rate of p-substituted phenols (k) increased with the increase of initial surface concentration gamma (representing the adsorption capacity of phenols to electrode surface), which indicated that organic compounds mainly reacted with adsorbed hydroxyl radicals at PbO2 anodes. For Ti/SnO2-Sb electrodes, however, k increased with the increase of the integrated parameter S (representing the effects of both sigma and gamma), which implied that organic compounds reacted with both adsorbed hydroxyl radicals and free hydroxyl radicals at SnO2 anodes.

Essential explanation of the strong mineralization performance of boron-doped diamond electrodes / Environ. Sci. Technol.

Subcellular mechanism of microbial inactivation during water disinfection by cold atmospheric-pressure plasma.

Subcellular mechanism of Escherichia coli inactivation during electrochemical disinfection with boron-doped diamond anode: A comparative study of three electrolytes

Previous studies in human patients and rodent models of Parkinson’s disease (PD) have established neuroprotection of dopaminergic (DA) neurons in substantia nigra pars compacta (SNC) by physical exercise, but the precise origin of this neuroprotective effect has yet to be elucidated. In this study, we tested a hypothesis that enhanced activity of DA neurons in SNC results in neuroprotection using the unilateral 6-hydroxydopamine (6-OHDA) injection model in mice. To increase activity of DA neurons chronically and specifically, we injected an adeno-associated viral vector carrying a step-function luminopsin (SFL) – a fusion protein of light-emitting Gaussia luciferase and light-sensing step-function channelrhodopsin 2 – into SNC ipsilateral to 6-OHDA using the pan-neuronal human synapsin I promoter or the Cre-lox system in transgenic mice expressing the recombinase under control of the tyrosine hydroxylase (TH) promoter. Upon application of SFL substrate, coelenterazine (CTZ), the luciferase moiety of luminopsin emits bioluminescence which in turn activate the opsin moiety. Daily injection of CTZ for 4 weeks ameliorated a stereotypical behavior, namely ipsiversive rotations, induced by unilateral 6-OHDA. In addition, postmortem immunohistochemistry against TH revealed less severe neurodegeneration of DA neurons compared to vehicle-injected control animals. Furthermore, when mice were pretreated with ANA-12, a selective antagonist for tropomyosin receptor kinase B (TrkB), the behavioral improvement and neuroprotective effect were diminished. These results suggest that increased neuronal activity of DA neurons provides neuroprotection against 6-OHDA injury and alleviates its symptoms through the brain-derived neurotrophic factor-TrkB pathway.

/pdf/chronic-activation-of-dopaminergic-neurons-via-2tc20g6w.pdf

Chronic activation of dopaminergic neurons via bioluminescence-optogenetics provides neuroprotection in a rodent model of Parkinson’s disease

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Subcellular mechanism of Escherichia coli inactivation during electrochemical disinfection with boron-doped diamond anode: A comparative study of three electrolytes

Chronic activation of dopaminergic neurons via bioluminescence-optogenetics provides neuroprotection in a rodent model of Parkinson’s disease