Zhong-Jing Wang
Huazhong University of Science and Technology
11 Papers
24 Citations
Zhong-Jing Wang is an academic researcher from Huazhong University of Science and Technology. The author has contributed to research in topics: Medicine & Internal medicine. The author has an hindex of 4, co-authored 8 publications. Previous affiliations of Zhong-Jing Wang include Technische Universität München.
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Papers
Decrease of global methylation improves significantly hepatic differentiation of Ad-MSCs: possible future application for urea detoxification.
Claudine Seeliger,Mihaela Culmes,Lilianna Schyschka,X Yan,X Yan,Georg Damm,Zhong-Jing Wang,Zhong-Jing Wang,Jörg Kleeff,Wolfgang E. Thasler,Jan G. Hengstler,Ulrich Stöckle,Sabrina Ehnert,Andreas K. Nussler,Andreas K. Nussler +14 more
TL;DR: Epigenetic changes in hepatocyte-like cells were induced by 5-azacytidine, which, in combination with other supplements, leads to significantly improved metabolic and enzymatic activities compared to nontreated cells, suggesting that these cells may be suitable for clinical application, especially for treatment of urea cycle disorders.
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Autologous Bone Marrow Mononuclear Cell Transplantation Therapy Improved Symptoms in Patients with Refractory Diabetic Sensorimotor Polyneuropathy via the Mechanisms of Paracrine and Immunomodulation: A Controlled Study.
Wei Wei,Li Li,Lin Deng,Zhong-Jing Wang,Jing-Jian Dong,Xiao-Yu Lyu,Ting Jia,Li Wang,Hong-Xiang Wang,Hong Mao,Shi Zhao +10 more
TL;DR: It is indicated that BM-MNCs transplantation is an effective and promising therapeutic strategy to treat refractory DSPN and the number of applied CD34+ cells and the duration of diabetes were the independent predictors of responding to the treatment.
11
Increased BMR in overweight and obese patients with type 2 diabetes may result from an increased fat-free mass.
TL;DR: It was concluded that overweight and obese Chinese adults with type 2 DM had increased BMRs compared with normal-weight controls, which may result from the difference in fat-free mass.
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Purα Repaired Expanded Hexanucleotide GGGGCC Repeat Noncoding RNA-Caused Neuronal Toxicity in Neuro-2a Cells
TL;DR: It is suggested that Purα inhibits expanded GGGGCC repeat-induced neuronal cell toxicity in Neuro-2a cells, which might reveal a novel mechanism of neurodegenerative diseases ALS and FTD.
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Association of height with peripheral arterial disease in type 2 diabetes.
TL;DR: A short stature seems to be associated with higher risk of PAD in Chinese diabetic patients, however, the cross-sectional nature of the study limits conclusions regarding the direction or causality.