Zefen Wang
Wuhan University
6 Papers
2 Citations
Zefen Wang is an academic researcher from Wuhan University. The author has contributed to research in topics: Medicine & Biology. The author has an hindex of 1, co-authored 2 publications.
Chat about Author
Papers
Advances in the Immunotherapeutic Potential of Isocitrate Dehydrogenase Mutations in Glioma
Fen Tang,Zhiyong Fan,Yian Wang,Tian Lan,Mengyue Wang,Fengliang Li,Wei Quan,Zhen-Xian Liu,Zefen Wang,Zhi-Qiang Li +9 more
TL;DR: Current knowledge about the effects of IDH mutations as well as other gene mutations on the immune microenvironment of gliomas are summarized and recent preclinical and clinical data related to IDH-mutant inhibitors for the treatment of gluomas are described.
14
Views on Careers in Clinical Neurosciences Among Neurosurgeons and Neurologists in China.
Rimas V. Lukas,Chao Ma,Jingcao Chen,Hongmei Dong,Jinxin Li,Zefen Wang,Ivy Jiang,Kai Ming Fu,Samita Satnarayan,Dara V.F. Albert,Renslow Sherer +10 more
TL;DR: A clearer understanding is provided of factors influencing career choice among clinical neuroscientists in China and differences with trainees elsewhere in the world emerge when evaluating additional factors influencing trainees pursuing careers in the clinical neurosciences.
2
Perceptions of clinical neurosciences among trainees in Wuhan, China
TL;DR: An understanding of attitudes toward neurology among medical students in China can play an important step in curriculum development and reform in the neurosciences.
2
In Regard to Chen et al: Could GBM Cell Growth Be Suppressed by Both Palmitoylation Inhibitor and Depalmitoylation Inhibitor?
TL;DR: Chen et al. as discussed by the authors found that SEDT2 protein mediated by ZDHHC16 is crucial for maintaining SEDTs stability, which, in turn, regulates DNA damage response via H3K36 trimethylation.
Current knowledge of protein palmitoylation in gliomas
TL;DR: This review focused on summarizing roles and relevant mechanisms of protein palmitoylational modification in gliomas, and found that palMIToylular modification is essential for remaining intracellular homeostasis and cell survival.