Yuexia Ding
Qingdao University
6 Papers
Yuexia Ding is an academic researcher from Qingdao University. The author has contributed to research in topics: Traumatic brain injury & Morris water navigation task. The author has an hindex of 3, co-authored 6 publications.
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Papers
Inhibition of microRNA-203 protects against traumatic brain injury induced neural damages via suppressing neuronal apoptosis and dementia-related molecues.
TL;DR: It is hypothesized that miR-203 inhibitor protects against neuronal damage and behavioral deficits by inhibition of Tau phosphorylation, ApoE4 expression and apoptosis, and rescue TBI mediated hippocampal LTP deficits and hippocampus dependent learning and memory dysfunction.
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Docosahexaenoic acid ameliorates traumatic brain injury involving JNK-mediated Tau phosphorylation signaling.
TL;DR: DHA suppressed TBI induced JNK and Tau hyperphosphorylation, rescued TBI mediated hippocampal LTP deficits and hippocampus dependent learning and memory dysfunction, and improved motor function.
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Docosahexaenoic Acid (DHA) Provides Neuroprotection in Traumatic Brain Injury Models via Activating Nrf2-ARE Signaling.
TL;DR: Investigation of the neuroprotective effects of docosahexaenoic acid in traumatic brain injury (TBI) models revealed that DHA facilitated the translocation of Nrf2 to the nucleus, potentially through activating the NRF2- ARE pathway.
miR-146a Mimics Ameliorates Traumatic Brain Injury Involving JNK and NF-κB Signaling Pathway.
TL;DR: In this paper, the effect of miR-146a mimic on TBI-induced neural damages was demonstrated by cortical impact injury (CCI), and the chemokine levels were examined by ELISA assays.
Edaravone attenuates neuronal apoptosis in hippocampus of rat traumatic brain injury model via activation of BDNF/TrkB signaling pathway
TL;DR: The study showed that administration with edaravone was able to inhibit neuronal apoptosis in the hippocampus in a rat TBI model, suggesting that the neuroprotective function of edarvone may relate to modulation of the BDNF/TrkB signaling pathway.