Yu Mu
Zhengzhou University
5 Papers
Yu Mu is an academic researcher from Zhengzhou University. The author has contributed to research in topics: Microvesicles & Metastasis. The author has an hindex of 4, co-authored 5 publications.
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Papers
Bone marrow mesenchymal stem cells-derived exosomal microRNA-193a reduces cisplatin resistance of non-small cell lung cancer cells via targeting LRRC1
Hongbo Wu,Xiaoqian Mu,Lei Liu,Huijuan Wu,Xiufeng Hu,Lijuan Chen,Jie Liu,Yu Mu,Fangfang Yuan,Wenjing Liu,Yanqiu Zhao +10 more
TL;DR: Functional studies report that BMSC-Exo shuffle miR-193a to suppress the colony formation, invasion, migration, and proliferation as well as advance apoptosis of NSCLC DDP-resistant cells via downregulating LRRC1.
LncRNA TDRG1 promotes the metastasis of NSCLC cell through regulating miR‐873‐5p/ZEB1 axis
TL;DR: It is demonstrated that TDRG1 is significantly overregulated in NSCLC tissues and cell lines and its expression is positively associated with the target of miR-873-5p, zinc finger e-box binding homeobox 1 (ZEB1) and positively regulates the expression of ZEB1.
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A silica–polymer composite nano system for tumor-targeted imaging and p53 gene therapy of lung cancer
Hongbo Wu,Yan-Qiu Zhao,Xiaoqian Mu,Huijuan Wu,Lijuan Chen,Wenjing Liu,Yu Mu,Jie Liu,Xudong Wei +8 more
TL;DR: In vivo Imaging and tumor targetability assays demonstrated that MB-NSi–p53–CS ternary complexes were a preferable system with desirable imaging and tumor-targeting properties.
Glypican-1-targeted and gemcitabine-loaded liposomes enhance tumor-suppressing effect on pancreatic cancer
Yu Mu,Dezhi Wang,Liangyu Bie,Suxia Luo,Xiaoqian Mu,Yanqiu Zhao +5 more
- 09 Oct 2020
TL;DR: Findings provided more insights into the anti-tumor potential for the biomedical application of GPC1-LP (GEM) in PDAC.
Exosomes Derived from Hypoxic Colorectal Cancer Cells Transfer miR-410-3p to Regulate Tumor Progression.
Xiufeng Hu,Yu Mu,Jie Liu,Xiaoqian Mu,Fangfang Gao,Lijuan Chen,Huijuan Wu,Hongbo Wu,Wenjing Liu,Yanqiu Zhao +9 more
TL;DR: The findings indicate that the hypoxic microenvironment in CRC may promote tumor cells to release miR-410-3p-rich exosomes that are transferred to normoxic cells to enhance tumor progression, revealing a new investigation into the therapeutic targets of exosome for CRC treatment.