Yikun Qu
Jiamusi University
10 Papers
57 Citations
Yikun Qu is an academic researcher from Jiamusi University. The author has contributed to research in topics: Medicine & Biology. The author has an hindex of 6, co-authored 7 publications.
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Papers
Effects of the HIF-1α and NF-κB loop on epithelial‑mesenchymal transition and chemoresistance induced by hypoxia in pancreatic cancer cells
Zhuoxin Cheng,Dawei Wang,Tao Liu,Weixin Liu,Weibin Xia,Jian Xu,Ying-Hai Zhang,Yikun Qu,Lin-Qi Guo,Long Ding,Jie Hou,Zhao-Hua Zhong +11 more
TL;DR: Molecular evidence is provided showing that the activation of the Hif-1α and NF-κB loop is mechanistically linked with the chemoresistance phenotype (EMT phenotype) of pancreatic cancer cells under hypoxic conditions, suggesting that the inactivation of HIF-1 α and NF -κB signaling by novel strategies may be a potential targeted therapeutic approach for overcoming EMT and Chemoresistance induced by hypoxia.
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Management of Postoperative Complications Following Splenectomy
TL;DR: The cause, characteristics, management, and outcome in patients with post-splenectomy hemorrhage were analyzed and early massive intraperitoneal hemorrhage is often preceded by earlier sentinel bleeding; careful clinical inquiry and ultrasonography are the mainstays of early diagnosis.
Long-term outcomes of laparoscopic splenectomy versus open splenectomy for idiopathic thrombocytopenic purpura.
TL;DR: The long-term outcome of laparoscopic splenectomy is not different from that of opensplenectomy for patients with ITP, and there was no significant difference in the relapse-free survival rate between the groups.
circRNA‑CER mediates malignant progression of breast cancer through targeting the miR‑136/MMP13 axis.
TL;DR: The present results suggested that circRNA‑CER may serve an important role in the progression of breast cancer by regulating the activity of the miR‑136/MMP13 axis, and may be a potential biomarker for the prediction and treatment of Breast cancer.
Downregulation of TBC1 Domain Family Member 24 (BC1D24) Inhibits Breast Carcinoma Growth via IGF1R/PI3K/AKT Pathway.
TL;DR: Silencing TBC1D24 inhibited the proliferation, migration, and invasion ability of MCF-7 cells, and promoted breast carcinoma growth through the IGF1R/PI3K/AKT pathway.