15 Papers
53 Citations
Yi Mao is an academic researcher from Huazhong University of Science and Technology. The author has contributed to research in topics: Medicine & Apolipoprotein E. The author has an hindex of 10, co-authored 13 publications.
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Papers
Digoxin reduces atherosclerosis in apolipoprotein E‐deficient mice
Huairui Shi,Xiaobo Mao,Yucheng Zhong,Yuzhou Liu,Xiaoqi Zhao,Kunwu Yu,Ruirui Zhu,Yuzhen Wei,Jianghao Zhu,Haitao Sun,Yi Mao,Qiutang Zeng +11 more
TL;DR: The present study was conducted to determine whether digoxin affects the development of atherosclerosis in a murine model of Atherosclerotic disease.
46
Thymic Stromal Lymphopoietin Attenuates the Development of Atherosclerosis in ApoE−/− Mice
Kunwu Yu,Pengfei Zhu,Qian Dong,Yucheng Zhong,Zhengfeng Zhu,Yingzhong Lin,Ying Huang,Kai Meng,Qingwei Ji,Guiwen Yi,Wei Zhang,Bangwei Wu,Yi Mao,Peng Cheng,Xiaoqi Zhao,Xiaobo Mao,Qiutang Zeng +16 more
TL;DR: A protective role for TSLP in Atherosclerosis is revealed that is possibly mediated by reestablishing a tolerogenic immune response, which may represent a novel possibility for treatment or prevention of atherosclerosis.
39
Total cholesterol content of erythrocyte membranes is associated with the severity of coronary artery disease and the therapeutic effect of rosuvastatin.
TL;DR: CEM levels are positively associated with the severity of CAD, meaning that CEM might contribute to the development of CAD and rosuvastatin could effectively help to attenuate the progression of CAD.
29
Overexpression of CXCL16 promotes a vulnerable plaque phenotype in Apolipoprotein E–Knockout Mice
Guiwen Yi,Qiutang Zeng,Xiaobo Mao,Min Cheng,Xiao-fang Yang,Hong-tao Liu,Yi Mao,Min Guo,Qin-wei Ji,Yucheng Zhong +9 more
TL;DR: Systemic CXCL16 becomes much higher in atherosclerosis, and it could be a potential atherogenic biomarker.
19
Lanatoside C Promotes Foam Cell Formation and Atherosclerosis.
Huairui Shi,Xiaobo Mao,Yucheng Zhong,Yuzhou Liu,Xiaoqi Zhao,Kunwu Yu,Ruirui Zhu,Yuzhen Wei,Jianghao Zhu,Haitao Sun,Yi Mao,Qiutang Zeng +11 more
TL;DR: It is demonstrated that lanatoside C aggravates the development of atherosclerosis by inducing PPARβ/δ expression, which mediates upregulation of SR-A and CD36, and promotes oxLDL uptake and foam-cell formation.