Yasuni Nakanuma
Kanazawa University
846 Papers
8.4K Citations
Yasuni Nakanuma is an academic researcher from Kanazawa University. The author has contributed to research in topics: Primary biliary cirrhosis & Bile duct. The author has an hindex of 94, co-authored 828 publications. Previous affiliations of Yasuni Nakanuma include Nagasaki University.
Chat about Author
Papers
Diffuse biliary tract involvement mimicking primary sclerosing cholangitis in an experimental model of chronic graft‐versus‐host disease in mice
TL;DR: The results suggest that PSC lesions might develop as a result of chronic cellular Immunologic mechanisms In GVHD across minor histocompatibility barriers.
25
Small hepatocellular carcinoma containing many Mallory bodies.
Yasuni Nakanuma,Goroku Ohta +1 more
TL;DR: The possibility arises that the process by which MBs are formed is a fundamental pathway that on some occasions may be linked with neoplastic transformation.
25
Analytical histopathological diagnosis of small hepatocellular nodules in chronic liver diseases
TL;DR: These low-grade HCC and dysplastic nodules should also be distinguished from classical HCC as well as large-sized regenerative nodules, which may augur a new horizon in the study of hepatocellular neoplasm.
Intrahepatic cholangiocarcinomas associated with nonbiliary cirrhosis. A clinicopathologic study.
Tadashi Terada,Tetsuji Kida,Yasuni Nakanuma,Hiroshi Kurumaya,Kenji Doishita,Nobutatsu Takayanagi +5 more
TL;DR: Clinically, patients with CC and cirrhosis were characterized by male preponderance, lower age, past history of liver injury, and elevated values of zinc sulfate and thymol turbidity tests, suggesting that the CCs are not hepatocellular neoplasms but true CCs.
25
Hilar cholangiocarcinoma is pathologically similar to pancreatic duct adenocarcinoma: suggestions of similar background and development
Yasuni Nakanuma,Yasunori Sato +1 more
TL;DR: Taken together, hilar CCA may differ from ICCA and CCA components of cHC‐CCA but have a similar development to PDAC, which may be explained by the unique anatomical, embryological and reactive nature of the pancreatobiliary tract.
24