8 Papers
62 Citations
Yanxia Lei is an academic researcher from National Health and Family Planning Commission. The author has contributed to research in topics: Kashin–Beck disease & Disease. The author has an hindex of 6, co-authored 8 publications. Previous affiliations of Yanxia Lei include Xi'an Jiaotong University.
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Papers
Is It the Appropriate Time to Stop Applying Selenium Enriched Salt in Kashin-Beck Disease Areas in China?
TL;DR: Gender, living area, Se intake without supplements, Se-enriched salt, oil source and protein intake were identified as significant factors of hair Se contents and Balanced dietary structure without Se supplement could effectively enhance and maintain children’s Se nutrition.
Nutrients Other than Selenium Are Important for Promoting Children’s Health in Kashin-Beck Disease Areas
Yujie Ning,Xi Wang,Xi Wang,Xiong Guo,Pan Zhang,Pengfei Qu,Feng Zhang,Sen Wang,Yanxia Lei,Mikko J. Lammi,Mikko J. Lammi +10 more
TL;DR: Children in Se-supplemented KBD-endemic areas have an insufficient intake of multiple nutrients, and school breakfast and lunch programmes are recommended, and strict implementation is the key to ensuring a positive effect.
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Comparing gene expression profiles of Kashin-Beck and Keshan diseases occurring within the same endemic areas of China.
Xi Wang,Shuang Wang,Shulan He,Feng Zhang,Wuhong Tan,Yanxia Lei,Hanjie Yu,Zheng Li,Yujie Ning,Youzhang Xiang,Xiong Guo +10 more
TL;DR: These shared differentially expressed genes may contribute to the distinct organ lesions, caused by common environmental risk factors of Kashin-Beck disease and Keshan disease.
Gene expression analysis suggests bone development-related genes GDF5 and DIO2 are involved in the development of Kashin-Beck disease in children rather than adults.
Yan Wen,Feng Zhang,Chunyan Li,Shulan He,Wuhong Tan,Yanxia Lei,Qiang Zhang,Hanjie Yu,Jingjing Zheng,Xiong Guo +9 more
TL;DR: Significant differences in gene expression pattern were identified between KBD children and KBD adults, indicating different molecular mechanisms underlying cartilage lesions of KBDchildren and adults with Kashin-Beck disease.
Mitochondrial-related gene expression profiles suggest an important role of PGC-1alpha in the compensatory mechanism of endemic dilated cardiomyopathy
Shulan He,Wuhong Tan,Zeng-Tie Zhang,Feng Zhang,Chengjuan Qu,Yanxia Lei,Yan-He Zhu,Hanjie Yu,Youzhang Xiang,Xiong Guo +9 more
TL;DR: It is suggested that PGC-1alpha regulated energy metabolism and anti-apoptosis might play an important role in the compensatory mechanism of KD.
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