Yangyang Sun
Soochow University (Suzhou)
7 Papers
16 Citations
Yangyang Sun is an academic researcher from Soochow University (Suzhou). The author has contributed to research in topics: Renal function & Creatinine. The author has an hindex of 2, co-authored 4 publications.
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Papers
m6A RNA modification modulates gene expression and cancer-related pathways in clear cell renal cell carcinoma.
TL;DR: This study presented the first m6A transcriptome-wide map of human ccRCC, which may shed lights on possible mechanisms of m 6A-mediated gene expression regulation.
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A circulating exosomal microRNA panel as a novel biomarker for monitoring post‐transplant renal graft function
TL;DR: A unique circulating exosomal miRNA panel was identified as an effective biomarker for monitoring post‐transplant renal graft function in this study and one‐year follow‐up revealed a stable recovery of allograft function in subjects with low calculated score from three‐miRNA panel.
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Quantitative increase in short-chain fatty acids, especially butyrate protects kidney from ischemia/reperfusion injury
TL;DR: In this paper, the authors explored butyrate effect on renal I/R injury and short-chain fatty acid (SCFAs) changes in renal transplant in male Sprague-Dawley rats.
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Let-7c-5p Is Involved in Chronic Kidney Disease by Targeting TGF-β Signaling.
TL;DR: Changes in hsa-let-7c-5p expression and its target gene TGF-β are associated with the disease status of CKD, which shows a negative correlation with proteinuria and creatinine levels, and a positive correlation with estimated glomerular filtration rate (eGFR).
CCL5 promotes the proliferation and metastasis of bladder cancer via the JAK2/STAT3 signaling pathway
Jie Shen,Cheng Chen,Zhen Chen,Pengfeng Gong,Lui Shiong Lee,Benjamin N. Schmeusser,Qianfeng Zhuang,Yangyang Sun,Dong Xue,Xiaozhou He +9 more
TL;DR: It is shown that CCL5 promotes proliferative, migratory, and tumor-growing BC cells in vitro and tumor metastasizing BC cells in vivo and the tumor-promotive role of CCL5 is dependent on activation of the JAK2/STAT3 signaling pathway.
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