10 Papers
103 Citations
Yan Ma is an academic researcher from Huazhong University of Science and Technology. The author has contributed to research in topics: Phosphorylation & GSK-3. The author has an hindex of 6, co-authored 9 publications.
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Papers
IQGAP1 activates Tcf signal independent of Rac1 and Cdc42 in injury and repair of bronchial epithelial cells
TL;DR: It is suggested that IQGAP1 mediated cell proliferation through activating Tcf in a manner independent of Rac1 and Cdc42 in wound repair of BECs.
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Cigarette smoke extract induces activation of β-catenin/TCF signaling through inhibiting GSK3β in human alveolar epithelial cell line
TL;DR: It is suggested that CSE induces the activation of beta-catenin/TCF signaling through inhibiting GSK3beta, implying a possible mechanism responsible for the injurious and cytotoxic effects on alveolar epithelial cell caused by cigarette smoke.
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Glycogen synthase kinase 3β and β-catenin are involved in the injury and repair of bronchial epithelial cells induced by scratching
TL;DR: The studies suggest that the scratching-induced injury and repair of BECs may involve inhibition of GSK3β activity which can lead to activation of the downstream signaling through β-catenin, providing a possible mechanism implicated in the injury and Repair of airway epithelium.
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Bleomycin-induced nuclear factor-κB activation in human bronchial epithelial cells involves the phosphorylation of glycogen synthase kinase 3β
TL;DR: The results suggest that BLM can induce the activation ofNF-kappaB signaling in BECs and this process is tightly associated with the phosphorylation status of GSK3beta, implying a possible regulatory mechanism of NF-kappB signaling during the toxic lung injury induced by BLM.
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IQGAP1 promotes cell proliferation and is involved in a phosphorylation-dependent manner in wound closure of bronchial epithelial cells.
TL;DR: The results suggest that IQGAP1 promotes cell proliferation and phosphorylation of IQG AP1 is involved in the process of wound closure in BECs.