Y. Wang
Duke University
8 Papers
384 Citations
Y. Wang is an academic researcher from Duke University. The author has contributed to research in topics: Dopamine receptor & Dopamine receptor D3. The author has an hindex of 8, co-authored 8 publications. Previous affiliations of Y. Wang include Howard Hughes Medical Institute.
Chat about Author
Papers
Autocrine signaling through ATP release represents a novel mechanism for cell volume regulation
TL;DR: Findings indicate that increases in cell volume lead to efflux of ATP through opening of a conductive pathway consistent with a channel, and that extracellular ATP is required for recovery from swelling.
336
Differential quantal release of histamine and 5-hydroxytryptamine from mast cells of vesicular monoamine transporter 2 knockout mice.
TL;DR: Although vesicular transport is the limiting factor determining quantal size of 5-HT and histamine release, intragranular association with the heparin matrix also plays a significant role.
89
Neural network of structures in which GABAB receptors regulate absence seizures in the lethargic (lh/lh) mouse model
TL;DR: This study found that anterior ventral lateral thalamic nucleus (VLa), nucleus reticularis thalami (NRT), nucleus reuniens (RE) passed all three screens, and hence are members of the neural network in which GABAB receptors regulate absence seizures in lh/lh mice.
60
Characterization of a swelling-activated anion conductance in homozygous typing cell hepatoma cells.
TL;DR: It is demonstrated that the HTC membrane anion permeability is closely coupled to changes in cell volume, and that the recovery from swelling depends upon activation of anion‐selective conductance pathways permeable to both Cl− and taurine.
31
Clozapine-, but not haloperidol-, induced increases in ΔFosB-like immunoreactivity are completely blocked in the striatum of mice lacking D3 dopamine receptors
George S. Robertson,Christopher J. Lee,Kavita Sridhar,Yusaku Nakabeppu,Michael Cheng,Y. Wang,Marc G. Caron +6 more
TL;DR: It is indicated that D3 receptor blockade is essential for clozapine‐induced increases in striatal ΔFosB‐Ir, and it is suggested that D 3 receptor antagonism may contribute to the unique therapeutic profile of this atypical antipsychotic.
27