Xiaoli Wei
Anhui Medical University
11 Papers
2 Citations
Xiaoli Wei is an academic researcher from Anhui Medical University. The author has contributed to research in topics: Medicine & Internal medicine. The author has an hindex of 3, co-authored 5 publications.
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Papers
Diagnostic, Therapeutic Predictive, and Prognostic Value of Neutrophil Extracellular Traps in Patients With Gastric Adenocarcinoma.
Yiyin Zhang,Yangyang Hu,Cui Ma,Hua Sun,Xiaoli Wei,Min Li,Wei Wei,Fei Zhang,Feng Yang,Hua Wang,Kangsheng Gu +10 more
TL;DR: The formation of NETs was discovered in the tissue microenvironment and PB of GC patients and the level ofNETs in the PB was a unfavorable independent prognostic factor for PFS in patients with advanced GC who had received advanced first-line treatment.
MiR-192-5p reverses cisplatin resistance by targeting ERCC3 and ERCC4 in SGC7901/DDP cells.
Xiaoque Xie,Nana Huang,Yiyin Zhang,Xiaoli Wei,Mengru Gao,Min Li,Jie Ning,Wei Liu,Qihong Zhao,Hua Wang,Kangsheng Gu +10 more
TL;DR: MiR-192-5p partially reversed GC cisplatin resistance by targeting ERCC3 and ERCC4, which participate in the NER pathway, suggesting that miR- 192-5P may be a potential biomarker and therapeutic target for GC cisPlatin resistance.
The regulatory mechanism of neutrophil extracellular traps in cancer biological behavior
TL;DR: In this article, the authors highlight how neutrophil extracellular traps (NETs) which are stimulated by various stimuli and signaling pathways, affects cancer biological behaviors via NETs.
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MicroRNA-362-5p enhances the cisplatin sensitivity of gastric cancer cells by targeting suppressor of zeste 12 protein.
Xiaoli Wei,Mengru Gao,Yaser Ahmed,Min Gao,Wen‑Bo Liu,Yiyin Zhang,Xiaoque Xie,Qihong Zhao,Hua Wang,Kangsheng Gu +9 more
TL;DR: The results of the present study suggested that dysregulated miR-362-5p may target SUZ12 to promote the development of cisplatin resistance and attenuate cisPlatin-induced apoptosis and may serve as a promising therapeutic strategy for patients with cis platin-resistant GC.
G protein-coupled receptor 35 attenuates nonalcoholic steatohepatitis by reprogramming cholesterol homeostasis in hepatocytes
Xiaoli Wei,Fan Yin,Miaomiao Wu,Qianqian Xie,Xueqin Zhao,Cheng Zhu,Ruiqian Xie,Chongqing Chen,Menghua Liu,Xue-Ying Wang,Ruixue Ren,Guijie Kang,Chenwen Zhu,Jingjing Cong,Hua Wang,Xuefu Wang +15 more
TL;DR: In this paper , G protein-coupled receptor 35 (GPR35) is involved in metabolic stresses, but its role in NAFLD is unknown, but hepatocyte GPR35 mitigates NASH by regulating hepatic cholesterol homeostasis.
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