Xiaojing Li
Northeast Agricultural University
26 Papers
Xiaojing Li is an academic researcher from Northeast Agricultural University. The author has contributed to research in topics: Apoptosis & Chemistry. The author has an hindex of 11, co-authored 17 publications.
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Papers
Effects of selenium-lead interaction on the gene expression of inflammatory factors and selenoproteins in chicken neutrophils.
Xiaojing Li,Mengyuan Xing,Menghao Chen,Jinxin Zhao,Ruifeng Fan,Xia Zhao,Changyu Cao,Jie Yang,Ziwei Zhang,Shiwen Xu +9 more
TL;DR: Se supplements could reduce Pb concentration in serum, had a mitigative effect on the activation of the NF-κB pathway and further enhanced the upward trend of selenoprotein expression induced by Pb exposure.
114
Cadmium induces BNIP3-dependent autophagy in chicken spleen by modulating miR-33-AMPK axis.
TL;DR: It is concluded that Cd induced the deregulation of miR-33-AMPK axis led to BNIP3-dependent autophagy in chicken spleen through AKT/mTOR and HSP70-NF-κB/JNK signal pathways and in-addition Cd could affect ion homeostasis inChicken spleen.
107
Hydrogen sulfide exposure triggers chicken trachea inflammatory injury through oxidative stress-mediated FOS/IL8 signaling.
TL;DR: H2S induced oxidative stress leads to tracheal inflammation through FOS/IL8 signaling, leading to excessive mucus secretion and absence of cilia, and new insights are provided for unveiling the biological effects of H2S in vivo and in vitro.
101
Selenomethionine alleviates LPS-induced chicken myocardial inflammation by regulating the miR-128-3p-p38 MAPK axis and oxidative stress
TL;DR: In this article, the effect of selenium on LPS-induced myocardial inflammation in chickens was explored, and the results showed that LPS stimulation inhibited miR-128-3p, which increased the expression of p38MAPK and NF-κB.
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Hydrogen sulfide exposure induces apoptosis and necroptosis through lncRNA3037/miR-15a/BCL2-A20 signaling in broiler trachea.
TL;DR: H2S exposure mediates apoptosis and necroptosis through lncRNA3037/miR-15/A20-BCL2 and negatively regulates each other, which provide new insights for unveiling the biological effects of H2S in vivo and in vitro.
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