Xiaohong Liu
Sichuan University
5 Papers
Xiaohong Liu is an academic researcher from Sichuan University. The author has contributed to research in topics: Lipotoxicity & Internal medicine. The author has an hindex of 2, co-authored 5 publications.
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Papers
β cell aging and age-related diabetes.
Min Zhu,Xiaohong Liu,Wen Liu,Yanrong Lu,Jingqiu Cheng,Younan Chen +5 more
- 03 Mar 2021
TL;DR: In this paper, the role of pancreatic β cell senescence and systemic aging in the pathogenesis of type 2 diabetes in elderly people remain poorly understood, and the current findings and viewpoints focusing on β cell aging and the development of Type 2 diabetes are discussed.
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Mesenchymal stromal cells protect hepatocytes from lipotoxicity through alleviation of endoplasmic reticulum stress by restoring SERCA activity.
Linzhao Li,Linzhao Li,Xin Zeng,Zhenzhen Liu,Xuanming Chen,Lan Li,Ruixi Luo,Xiaohong Liu,Jie Zhang,Jingping Liu,Yanrong Lu,Jingqiu Cheng,Younan Chen +12 more
TL;DR: In this article, mesenchymal stromal cells (MSCs) modulate metabolic balance and attenuate hepatic lipotoxicity in the context of non-alcoholic fatty liver disease (NAFLD).
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Oleic acid protects insulin-secreting INS-1E cells against palmitic acid-induced lipotoxicity along with an amelioration of ER stress.
Xiaohong Liu,Xin Zeng,Xuanming Chen,Ruixi Luo,Linzhao Li,Chengshi Wang,Jingping Liu,Jingqiu Cheng,Yanrong Lu,Younan Chen +9 more
TL;DR: OA alleviated PA-induced lipotoxicity in INS-1E cells and improved insulin sensitivity in HFD rats and regulated the ER Ca2+ homeostasis, which may be responsible for its beneficial effects in β cells.
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Correction to: Oleic acid ameliorates palmitic acid induced hepatocellular lipotoxicity by inhibition of ER stress and pyroptosis.
Xin Zeng,Min Zhu,Xiaohong Liu,Xuanmin Chen,Yujia Yuan,Lan Li,Jingping Liu,Yanrong Lu,Jingqiu Cheng,Younan Chen +9 more
TL;DR: The original article possessed a minor typo in first author, Xin Zeng’s name which has since been corrected.
Mesenchymal stem cells alleviate palmitic acid-induced endothelial-to-mesenchymal transition by suppressing endoplasmic reticulum stress.
Ruixi Luo,Ruixi Luo,Linzhao Li,Xiaohong Liu,Yujia Yuan,Wuzheng Zhu,Lan Li,Jingping Liu,Yanrong Lu,Jingqiu Cheng,Younan Chen +10 more
TL;DR: Findings indicated that MSCs were able to attenuate endothelial lipotoxicity through inhibiting ER stress and EndMT, in which STC1 secreted from M SCs may play a critical role.