Xiaofan Su
Xi'an Jiaotong University
12 Papers
17 Citations
Xiaofan Su is an academic researcher from Xi'an Jiaotong University. The author has contributed to research in topics: Protein kinase B & Cancer. The author has an hindex of 7, co-authored 10 publications.
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Papers
Activation of AMPK inhibits PDGF-induced pulmonary arterial smooth muscle cells proliferation and its potential mechanisms
Yang Song,Yuanyuan Wu,Xiaofan Su,Yanting Zhu,Lu Liu,Yilin Pan,Bo Zhu,Lan Yang,Li Gao,Manxiang Li +9 more
TL;DR: The study suggests that the activation of AMPK negatively regulates mTOR activity to suppress PASMC proliferation and therefore has a potential value in the prevention and treatment of pulmonary hypertension by negatively modulating pulmonary vascular remodeling.
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Activation of AMPK α2 inhibits airway smooth muscle cells proliferation.
TL;DR: The study suggests that activation of AMPK, particularly AMPK α2, negatively regulates mTOR activity to suppress ASMCs proliferation and therefore has a potential value in the prevention and treatment of asthma by negatively modulating airway remodeling.
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Activation of peroxisome proliferation-activated receptor-γ inhibits transforming growth factor-β1-induced airway smooth muscle cell proliferation by suppressing Smad-miR-21 signaling.
Lu Liu,Yilin Pan,Cui Zhai,Yanting Zhu,Rui Ke,Wenhua Shi,Jian Wang,Xin Yan,Xiaofan Su,Yang Song,Li Gao,Manxiang Li +11 more
TL;DR: The study suggests that the activation of PPAR‐γ inhibits rat ASMC proliferation by suppressing Smad–miR‐21 signaling and therefore has a potential value in the prevention and treatment of asthma by negatively modulating airway remodeling.
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Association of TLR4 (896A/G and 1196C/T) Gene Polymorphisms with Asthma Risk: A Meta-Analysis.
TL;DR: It is suggested that T allele of the TLR4 +1196C/T might act as a protective factor against the development of asthma.
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•Journal Article
Association between IL-17F rs763780 polymorphism and susceptibility of asthma: a meta-analysis.
TL;DR: The present meta-analysis, combining all currently available data, suggests that IL-17F rs763780 polymorphism is not associated with the susceptibility of asthma.
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